Strict 3' splice site sequence requirements for U2 snRNP recruitment after U2AF binding underlie a genetic defect leading to autoimmune disease.
about
Isolated pseudo-RNA-recognition motifs of SR proteins can regulate splicing using a noncanonical mode of RNA recognitionAlternative splicing of RNA triplets is often regulated and accelerates proteome evolution.Intron retention in the alternatively spliced region of RON results from weak 3' splice site recognition.The Activation-Induced Assembly of an RNA/Protein Interactome Centered on the Splicing Factor U2AF2 Regulates Gene Expression in Human CD4 T CellsSplicing inhibition of U2AF65 leads to alternative exon skipping.Intronic features that determine the selection of the 3' splice site.Post-transcriptional coordination of immunological responses by RNA-binding proteins.Novel insights into FAS defects underlying autoimmune lymphoproliferative syndrome revealed by studies in consanguineous patients.
P2860
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P2860
Strict 3' splice site sequence requirements for U2 snRNP recruitment after U2AF binding underlie a genetic defect leading to autoimmune disease.
description
2011 nî lūn-bûn
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2011年の論文
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2011年論文
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2011年論文
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2011年論文
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2011年論文
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name
Strict 3' splice site sequence ...... leading to autoimmune disease.
@en
type
label
Strict 3' splice site sequence ...... leading to autoimmune disease.
@en
prefLabel
Strict 3' splice site sequence ...... leading to autoimmune disease.
@en
P2093
P2860
P356
P1433
P1476
Strict 3' splice site sequence ...... leading to autoimmune disease.
@en
P2093
Anna Corrionero
José María Izquierdo
Veronica A Raker
P2860
P304
P356
10.1261/RNA.2444811
P407
P577
2011-01-13T00:00:00Z