Is the inflammasome a potential therapeutic target in renal disease? - Wikidata - awgldk - TriplyDB

Is the inflammasome a potential therapeutic target in renal disease?

Is the inflammasome a potential therapeutic target in renal disease?

http://www.wikidata.org/entity/Q41934684

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The Nucleotide Oligomerization Domain-Like Receptors in Kidney Injury ROS-Mediated NLRP3 Inflammasome Activation in Brain, Heart, Kidney, and Testis Ischemia/Reperfusion Injury Secondary amyloidosis in autoinflammatory diseases and the role of inflammation in renal damage Nephropathy in dietary hyperoxaluria: A potentially preventable acute or chronic kidney disease Targeting HO-1 by Epigallocatechin-3-Gallate Reduces Contrast-Induced Renal Injury via Anti-Oxidative Stress and Anti-Inflammation Pathways.NLRP3 Inflammasome Polymorphism and Macrovascular Complications in Type 2 Diabetes Patients Renal systems biology of patients with systemic inflammatory response syndrome Characterization and Activation of NLRP3 Inflammasomes in the Renal Medulla in Mice.Transcriptome analysis reveals manifold mechanisms of cyst development in ADPKD.Influence of the Expression of Inflammatory Markers on Kidney after Fetal Programming in an Experimental Model of Renal Failure.Renal P2 receptors and hypertension.Inflammasome Activation in Chronic Glomerular Diseases.Immunity and inflammation in diabetic kidney disease: translating mechanisms to biomarkers and treatment targets.Efficacy and Pharmacology of the NLRP3 Inflammasome Inhibitor CP-456,773 (CRID3) in Murine Models of Dermal and Pulmonary Inflammation.Anti-inflammatory effects of triptolide on IgA nephropathy in rats.Pioglitazone ameliorates glomerular NLRP3 inflammasome activation in apolipoprotein E knockout mice with diabetes mellitus.Mitochondria Protection after Acute Ischemia Prevents Prolonged Upregulation of IL-1β and IL-18 and Arrests CKD.Transgenic expression of human APOL1 risk variants in podocytes induces kidney disease in mice.The regulation of NLRP3 inflammasome expression during the development of cardiac contractile dysfunction in chronic kidney disease.The study on the material basis and the mechanism for anti-renal interstitial fibrosis efficacy of rhubarb through integration of metabonomics and network pharmacology.Polydatin attenuates potassium oxonate-induced hyperuricemia and kidney inflammation by inhibiting NF-κB/NLRP3 inflammasome activation via the AMPK/SIRT1 pathway.Expression of TLR2, NOD1, and NOD2 and the NLRP3 Inflammasome in Renal Tubular Epithelial Cells of Male versus Female Mice.Regulation of TLR2 and NLRP3 in primary murine renal tubular epithelial cells.P2X7 receptor antagonism ameliorates renal dysfunction in a rat model of sepsis.Inflammation-Related Mechanisms in Chronic Kidney Disease Prediction, Progression, and Outcome

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P2860

Is the inflammasome a potential therapeutic target in renal disease?

http://www.wikidata.org/entity/Q41934684

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2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

Is the inflammasome a potential therapeutic target in renal disease?

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Is the inflammasome a potential therapeutic target in renal disease?

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Is the inflammasome a potential therapeutic target in renal disease?

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Is the inflammasome a potential therapeutic target in renal disease?

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Clare M Turner

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Frederick W K Tam

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Nishkantha Arulkumaran

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Robert J Unwin

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P2860

A pilot study of IL-1 inhibition by anakinra in acute gout

A novel role for interleukin-18 in adhesion molecule induction through NF kappa B and phosphatidylinositol (PI) 3-kinase-dependent signal transduction pathways

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Bcl-2 and Bcl-XL regulate proinflammatory caspase-1 activation by interaction with NALP1

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The CATERPILLER protein monarch-1 is an antagonist of toll-like receptor-, tumor necrosis factor alpha-, and Mycobacterium tuberculosis-induced pro-inflammatory signals

Innate immune activation through Nalp3 inflammasome sensing of asbestos and silica

Pyogenic bacterial infections in humans with MyD88 deficiency

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ATP binding by monarch-1/NLRP12 is critical for its inhibitory function

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