about
Glucocorticoid-induced tumor necrosis factor receptor family-related ligand triggering upregulates vascular cell adhesion molecule-1 and intercellular adhesion molecule-1 and promotes leukocyte adhesionA new member of the tumor necrosis factor/nerve growth factor receptor family inhibits T cell receptor-induced apoptosisGILZ as a Mediator of the Anti-Inflammatory Effects of GlucocorticoidsThe expanding role of immunopharmacology: IUPHAR Review 16Modulation of T-cell activation by the glucocorticoid-induced leucine zipper factor via inhibition of nuclear factor kappaBGITR interacts with the pro-apoptotic protein Siva and induces apoptosisA new dexamethasone-induced gene of the leucine zipper family protects T lymphocytes from TCR/CD3-activated cell deathNGF-promoted axon growth and target innervation requires GITRL-GITR signaling.Transcriptional regulation of kinases downstream of the T cell receptor: another immunomodulatory mechanism of glucocorticoids.Role of GITR in activation response of T lymphocytes.Synthesis of glucocorticoid-induced leucine zipper (GILZ) by macrophages: an anti-inflammatory and immunosuppressive mechanism shared by glucocorticoids and IL-10.Murine B cell development and antibody responses to model antigens are not impaired in the absence of the TNF receptor GITR.GITR, a member of the TNF receptor superfamily, is costimulatory to mouse T lymphocyte subpopulations.Expansion of regulatory GITR+CD25 low/-CD4+ T cells in systemic lupus erythematosus patients.Targeting glucocorticoid side effects: selective glucocorticoid receptor modulator or glucocorticoid-induced leucine zipper? A perspective.GITR/GITRL: more than an effector T cell co-stimulatory system.The glucocorticoid-induced TNF receptor family-related protein (GITR) is critical to the development of acute pancreatitis in mice.Molecular mechanisms of immunomodulatory activity of glucocorticoids.Lymphokine-activated killer cell activity Characteristics of effector cells and their progenitors in blood and spleen.Glucocorticoid-induced tumour necrosis factor receptor-related protein: a key marker of functional regulatory T cells.The viability of Lactobacillus fermentum CECT5716 is not essential to exert intestinal anti-inflammatory properties.Glucocorticoid-induced leucine zipper (GILZ) over-expression in T lymphocytes inhibits inflammation and tissue damage in spinal cord injury.GITR: a multifaceted regulator of immunity belonging to the tumor necrosis factor receptor superfamily.Pharmacological modulation of GITRL/GITR system: therapeutic perspectives.Modulation of acute and chronic inflammation of the lung by GITR and its ligand.Identification of regulatory T cells in systemic lupus erythematosus.Role of caspase-8 in thymus function.Overexpression of Glucocorticoid-induced Leucine Zipper (GILZ) increases susceptibility to Imiquimod-induced psoriasis and involves cutaneous activation of TGF-β1.Glucocorticoid-induced leucine zipper (GILZ): a new important mediator of glucocorticoid action.GITR: a modulator of immune response and inflammation.Balance between regulatory T and Th17 cells in systemic lupus erythematosus: the old and the new.Mechanisms of the anti-inflammatory effects of glucocorticoids: genomic and nongenomic interference with MAPK signaling pathways.Pharmacological modulation of caspase-8 in thymus-related medical conditions.GITR+ regulatory T cells in the treatment of autoimmune diseases.Role of regulatory T cells in rheumatoid arthritis: facts and hypothesis.The Hexane Fraction of Bursera microphylla A Gray Induces p21-Mediated Antiproliferative and Proapoptotic Effects in Human Cancer-Derived Cell Lines.The proinflammatory cytokine GITRL contributes to TRAIL-mediated neurotoxicity in the HCN-2 human neuronal cell line.Artesunate induces ROS- and p38 MAPK-mediated apoptosis and counteracts tumor growth in vivo in embryonal rhabdomyosarcoma cells.Glucocorticoid-induced leucine zipper: a critical factor in macrophage endotoxin tolerance.L-GILZ binds p53 and MDM2 and suppresses tumor growth through p53 activation in human cancer cells.
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Carlo Riccardi
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Carlo Riccardi
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Carlo Riccardi
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Carlo Riccardi
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Carlo Riccardi
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Carlo Riccardi
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Carlo Riccardi
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