Potassium does not mimic EDHF in rat mesenteric arteries.
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Endothelium-dependent smooth muscle hyperpolarization: do gap junctions provide a unifying hypothesis?Endothelium-derived hyperpolarizing factor and potassium use different mechanisms to induce relaxation of human subcutaneous resistance arteriesBlockade of chloride channels reveals relaxations of rat small mesenteric arteries to raised potassiumSuppression of K(+)-induced hyperpolarization by phenylephrine in rat mesenteric artery: relevance to studies of endothelium-derived hyperpolarizing factorEndothelium-dependent vasorelaxation independent of nitric oxide and K(+) release in isolated renal arteries of ratsCellular target of voltage and calcium-dependent K(+) channel blockers involved in EDHF-mediated responses in rat superior mesenteric arteryRole of the endothelium on arterial vasomotion.A comparison of responses to raised extracellular potassium and endothelium-derived hyperpolarizing factor (EDHF) in rat pressurised mesenteric arteries.EDHF: spreading the influence of the endothelium.Acetylcholine-induced vasodilation may depend entirely upon NO in the femoral artery of young pigletsOuabain exerts biphasic effects on connexin functionality and expression in vascular smooth muscle cellsVasorelaxation of goat mesenteric artery is mediated by endothelial Na(+)-K(+)-ATPase.A mathematical model of vasoreactivity in rat mesenteric arterioles: I. Myoendothelial communicationEDH: endothelium-dependent hyperpolarization and microvascular signalling.Potentiation of EDHF-mediated relaxation by chloride channel blockers.Reduced activity of SKC a and Na-K ATPase underlies the accelerated impairment of EDH-type relaxations in mesenteric arteries of aging spontaneously hypertensive rats.Extracellular Calcium-Dependent Modulation of Endothelium Relaxation in Rat Mesenteric Small Artery: The Role of Potassium Signaling.Role of Ba2+-resistant K+ channels in endothelium-dependent hyperpolarization of rat small mesenteric arteries.K+-induced hyperpolarization in rat mesenteric artery: identification, localization and role of Na+/K+-ATPases.The Na-K-ATPase is a target for an EDHF displaying characteristics similar to potassium ions in the porcine renal interlobar artery.Different pathways with distinct properties conduct dilations in the microcirculation in vivo.Properties of smooth muscle hyperpolarization and relaxation to K+ in the rat isolated mesenteric artery.Determinants of renal microvascular response to ACh: afferent and efferent arteriolar actions of EDHF.Propionate-induced relaxation in rat mesenteric arteries: a role for endothelium-derived hyperpolarising factor.Critical role of gap junctions in endothelium-dependent hyperpolarization in rat mesenteric arteries.K(Ca) channel blockers reveal hyperpolarization and relaxation to K+ in rat isolated mesenteric artery.EDHF-mediated vasodilation involves different mechanisms in normotensive and hypertensive rat lungs.Spreading dilatation in rat mesenteric arteries associated with calcium-independent endothelial cell hyperpolarization.Electrical coupling and release of K+ from endothelial cells co-mediate ACh-induced smooth muscle hyperpolarization in guinea-pig inner ear artery.Ouabain exerts biphasic effects on connexin functionality and expression in vascular smooth muscle cells.
P2860
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P2860
Potassium does not mimic EDHF in rat mesenteric arteries.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
Potassium does not mimic EDHF in rat mesenteric arteries.
@en
type
label
Potassium does not mimic EDHF in rat mesenteric arteries.
@en
prefLabel
Potassium does not mimic EDHF in rat mesenteric arteries.
@en
P2093
P2860
P356
P1476
Potassium does not mimic EDHF in rat mesenteric arteries.
@en
P2093
J M Doughty
P D Langton
P2860
P304
P356
10.1038/SJ.BJP.0703412
P407
P577
2000-07-01T00:00:00Z