Role of intracellular Ca2+ in stimulation-induced increases in transmitter release at the frog neuromuscular junction.
about
Evidence that mitochondria buffer physiological Ca2+ loads in lizard motor nerve terminals.Chronic intermittent hypoxia and hypercapnia inhibit the hypothalamic paraventricular nucleus neurotransmission to parasympathetic cardiac neurons in the brain stem.Mitochondrial Ca2+ uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals.Inhibition of mitochondrial Ca2+ uptake affects phasic release from motor terminals differently depending on external [Ca2+].A vagal nerve branch controls swallowing directly in the seawater eel.Long-term in vivo modulation of synaptic efficacy at the neuromuscular junction of Rana pipiens frogs.
P2860
Role of intracellular Ca2+ in stimulation-induced increases in transmitter release at the frog neuromuscular junction.
description
1994 nî lūn-bûn
@nan
1994年の論文
@ja
1994年論文
@yue
1994年論文
@zh-hant
1994年論文
@zh-hk
1994年論文
@zh-mo
1994年論文
@zh-tw
1994年论文
@wuu
1994年论文
@zh
1994年论文
@zh-cn
name
Role of intracellular Ca2+ in ...... e frog neuromuscular junction.
@en
type
label
Role of intracellular Ca2+ in ...... e frog neuromuscular junction.
@en
prefLabel
Role of intracellular Ca2+ in ...... e frog neuromuscular junction.
@en
P2093
P356
P1476
Role of intracellular Ca2+ in ...... e frog neuromuscular junction.
@en
P2093
P304
P356
10.1085/JGP.104.2.337
P577
1994-08-01T00:00:00Z