Macromitophagy, neutral lipids synthesis, and peroxisomal fatty acid oxidation protect yeast from "liponecrosis", a previously unknown form of programmed cell death
about
External and internal triggers of cell death in yeastA novel approach to the discovery of anti-tumor pharmaceuticals: searching for activators of liponecrosisAutophagy in the physiology and pathology of the central nervous systemCommunications between Mitochondria, the Nucleus, Vacuoles, Peroxisomes, the Endoplasmic Reticulum, the Plasma Membrane, Lipid Droplets, and the Cytosol during Yeast Chronological AgingDiscovery of plant extracts that greatly delay yeast chronological aging and have different effects on longevity-defining cellular processesMechanisms Underlying the Essential Role of Mitochondrial Membrane Lipids in Yeast Chronological AgingMechanisms underlying the anti-aging and anti-tumor effects of lithocholic bile acidEmpirical verification of evolutionary theories of aging.Quasi-programmed aging of budding yeast: a trade-off between programmed processes of cell proliferation, differentiation, stress response, survival and death defines yeast lifespan.Mechanisms by which different functional states of mitochondria define yeast longevity.Cell-autonomous mechanisms of chronological aging in the yeast Saccharomyces cerevisiaeYeast MRX deletions have short chronological life span and more triacylglycerols.Mechanism of liponecrosis, a distinct mode of programmed cell death.Another face of cell death.Lithocholic bile acid accumulated in yeast mitochondria orchestrates a development of an anti-aging cellular pattern by causing age-related changes in cellular proteome.Caloric restriction extends yeast chronological lifespan via a mechanism linking cellular aging to cell cycle regulation, maintenance of a quiescent state, entry into a non-quiescent state and survival in the non-quiescent state.Modulation of gluconeogenesis and lipid production in an engineered oleaginous Saccharomyces cerevisiae transformant.Some Metabolites Act as Second Messengers in Yeast Chronological Aging.Yeast Cells Exposed to Exogenous Palmitoleic Acid Either Adapt to Stress and Survive or Commit to Regulated Liponecrosis and Die.Caloric restriction delays yeast chronological aging by remodeling carbohydrate and lipid metabolism, altering peroxisomal and mitochondrial functionalities, and postponing the onsets of apoptotic and liponecrotic modes of regulated cell death.Mechanisms through which lithocholic acid delays yeast chronological aging under caloric restriction conditions
P2860
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P2860
Macromitophagy, neutral lipids synthesis, and peroxisomal fatty acid oxidation protect yeast from "liponecrosis", a previously unknown form of programmed cell death
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Macromitophagy, neutral lipids ...... form of programmed cell death
@en
type
label
Macromitophagy, neutral lipids ...... form of programmed cell death
@en
prefLabel
Macromitophagy, neutral lipids ...... form of programmed cell death
@en
P2093
P2860
P356
P1433
P1476
Macromitophagy, neutral lipids ...... form of programmed cell death
@en
P2093
Adam Beach
Amanda Piano
Anna Leonov
Hojatollah Vali
Leila Khelghatybana
Rachel Feldman
Sara Sheibani
Sevan Mattie
Vincent R Richard
Vladimir I Titorenko
P2860
P304
P356
10.4161/CC.26885
P577
2013-10-28T00:00:00Z