Complementation of aprataxin deficiency by base excision repair enzymes in mitochondrial extracts.

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Complementation of aprataxin deficiency by base excision repair enzymes in mitochondrial extracts.

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http://www.wikidata.org/entity/Q41961242

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

@yue

2017年論文

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2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

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2017年论文

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Complementation of aprataxin d ...... mes in mitochondrial extracts.

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Complementation of aprataxin d ...... mes in mitochondrial extracts.

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Complementation of aprataxin d ...... mes in mitochondrial extracts.

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P1476

Complementation of aprataxin d ...... ymes in mitochondrial extracts

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P2093

Hiroyuki Sasanuma

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Kei Kadoda

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Keizo Tano

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Masataka Tsuda

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Matthew J Longley

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Melike Çaglayan

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Rachel Krasich

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Rajendra Prasad

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Shunichi Takeda

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P2860

CK2 phosphorylation-dependent interaction between aprataxin and MDC1 in the DNA damage response

The mitochondrial p55 accessory subunit of human DNA polymerase gamma enhances DNA binding, promotes processive DNA synthesis, and confers N-ethylmaleimide resistance

The neurodegenerative disease protein aprataxin resolves abortive DNA ligation intermediates

The ataxia-oculomotor apraxia 1 gene product has a role distinct from ATM and interacts with the DNA strand break repair proteins XRCC1 and XRCC4

Aprataxin, the causative protein for EAOH is a nuclear protein with a potential role as a DNA repair protein

Cloning and characterization of the human mitochondrial DNA polymerase, DNA polymerase gamma

Aprataxin, a novel protein that protects against genotoxic stress

Multiplex genome engineering using CRISPR/Cas systems

Eukaryotic DNA ligases: structural and functional insights

Defective DNA ligation during short-patch single-strand break repair in ataxia oculomotor apraxia 1

P304

10079-10088

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P31

scholarly article

P356

10.1093/NAR/GKX654

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

William C Copeland

Samuel H. Wilson

P577

2017-09-01T00:00:00Z

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P698

28973450

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P932

5622373

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