Apn1 AP-endonuclease is essential for the repair of oxidatively damaged DNA bases in yeast frataxin-deficient cells.
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Frataxin Deficiency Promotes Excess Microglial DNA Damage and Inflammation that Is Rescued by PJ34Neurodegeneration in Friedreich's ataxia: from defective frataxin to oxidative stressFriedreich's ataxia, frataxin, PIP5K1B: echo of a distant fracas.Oxidative stress in inherited mitochondrial diseases.Frataxin deficiency leads to reduced expression and impaired translocation of NF-E2-related factor (Nrf2) in cultured motor neuronsFrataxin deficiency leads to defects in expression of antioxidants and Nrf2 expression in dorsal root ganglia of the Friedreich's ataxia YG8R mouse model
P2860
Apn1 AP-endonuclease is essential for the repair of oxidatively damaged DNA bases in yeast frataxin-deficient cells.
description
2012 nî lūn-bûn
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2012年の論文
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name
Apn1 AP-endonuclease is essent ...... east frataxin-deficient cells.
@en
type
label
Apn1 AP-endonuclease is essent ...... east frataxin-deficient cells.
@en
prefLabel
Apn1 AP-endonuclease is essent ...... east frataxin-deficient cells.
@en
P2093
P2860
P50
P356
P1476
Apn1 AP-endonuclease is essent ...... yeast frataxin-deficient cells
@en
P2093
Caroline Brossas
Françoise Auchère
Nicole Boggetto
P2860
P304
P356
10.1093/HMG/DDS230
P577
2012-06-16T00:00:00Z