Subacute zinc administration and L-NAME caused an increase of NO, zinc, lipoperoxidation, and caspase-3 during a cerebral hypoxia-ischemia process in the rat.
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Prophylactic Chronic Zinc Administration Increases Neuroinflammation in a Hypoxia-Ischemia ModelZinc: indications in brain disorders.Transient transfection of human CDNF gene reduces the 6-hydroxydopamine-induced neuroinflammation in the rat substantia nigraProphylactic Subacute Administration of Zinc Increases CCL2, CCR2, FGF2, and IGF-1 Expression and Prevents the Long-Term Memory Loss in a Rat Model of Cerebral Hypoxia-Ischemia.Dysregulation of zinc/lipid metabolism‑associated genes in the rat hippocampus and cerebral cortex in early adulthood following recurrent neonatal seizures.Prophylactic Zinc and Therapeutic Selenium Administration Increases the Antioxidant Enzyme Activity in the Rat Temporoparietal Cortex and Improves Memory after a Transient Hypoxia-Ischemia
P2860
Subacute zinc administration and L-NAME caused an increase of NO, zinc, lipoperoxidation, and caspase-3 during a cerebral hypoxia-ischemia process in the rat.
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2013 nî lūn-bûn
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
@zh-hk
2013年論文
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2013年論文
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2013年论文
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2013年论文
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2013年论文
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name
Subacute zinc administration a ...... a-ischemia process in the rat.
@en
type
label
Subacute zinc administration a ...... a-ischemia process in the rat.
@en
prefLabel
Subacute zinc administration a ...... a-ischemia process in the rat.
@en
P2093
P2860
P50
P356
P1476
Subacute zinc administration a ...... ia-ischemia process in the rat
@en
P2093
Bertha Alicia Leon-Chavez
Celia Piña-Leyva
Daniel Martinez-Fong
Eduardo Brambila
Hector Rubio
Jorge Cebada
Maricela Torres-Soto
María de Jesus Gomez-Villalobos
Patricia Lopez-Moreno
Victor Manuel Blanco-Alvarez
P2860
P304
P356
10.1155/2013/240560
P50
P577
2013-08-07T00:00:00Z