Free radical generation by neutrophils: a potential mechanism of cellular injury in acute alcoholic hepatitis.
about
Lipid peroxidation in rats chronically fed ethanolChronic ethanol exposure potentiates lipopolysaccharide liver injury despite inhibiting Jun N-terminal kinase and caspase 3 activation.Effects of beta-carotene on cell viability and antioxidant status of hepatocytes from chronically ethanol-fed rats.
P2860
Free radical generation by neutrophils: a potential mechanism of cellular injury in acute alcoholic hepatitis.
description
1987 nî lūn-bûn
@nan
1987年の論文
@ja
1987年論文
@yue
1987年論文
@zh-hant
1987年論文
@zh-hk
1987年論文
@zh-mo
1987年論文
@zh-tw
1987年论文
@wuu
1987年论文
@zh
1987年论文
@zh-cn
name
Free radical generation by neu ...... in acute alcoholic hepatitis.
@en
type
label
Free radical generation by neu ...... in acute alcoholic hepatitis.
@en
prefLabel
Free radical generation by neu ...... in acute alcoholic hepatitis.
@en
P2860
P356
P1433
P1476
Free radical generation by neu ...... in acute alcoholic hepatitis.
@en
P2093
P2860
P304
P356
10.1136/GUT.28.9.1157
P407
P577
1987-09-01T00:00:00Z