Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis. - Wikidata - awgldk - TriplyDB

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

http://www.wikidata.org/entity/Q42021296

about

Hematopoietic and mesenchymal stem cells for the treatment of chronic respiratory diseases: role of plasticity and heterogeneity Pleural mesothelial cell differentiation and invasion in fibrogenic lung injury Controversies over the Epithelial-to-Mesenchymal Transition in Liver Fibrosis Breakdown of Epithelial Barrier Integrity and Overdrive Activation of Alveolar Epithelial Cells in the Pathogenesis of Acute Respiratory Distress Syndrome and Lung Fibrosis Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosis EMT and interstitial lung disease: a mysterious relationship Epigenomics of idiopathic pulmonary fibrosis Myofibroblasts Molecular biomarkers in idiopathic pulmonary fibrosis DA-Raf-Mediated Suppression of the Ras--ERK Pathway Is Essential for TGF-β1-Induced Epithelial-Mesenchymal Transition in Alveolar Epithelial Type 2 Cells Role of CD248 as a potential severity marker in idiopathic pulmonary fibrosis.Pulmonary fibrosis: patterns and perpetrators Epithelial-mesenchymal transition: An emerging target in tissue fibrosis Integrated long non-coding RNA analyses identify novel regulators of epithelial-mesenchymal transition in the mouse model of pulmonary fibrosis Hypoxia-Induced Epithelial-Mesenchymal Transition Is Involved in Bleomycin-Induced Lung Fibrosis Multi-walled carbon nanotubes directly induce epithelial-mesenchymal transition in human bronchial epithelial cells via the TGF-β-mediated Akt/GSK-3β/SNAIL-1 signalling pathway Workshop on idiopathic pulmonary fibrosis in older adults Molecular analysis of idiopathic subglottic stenosis for Mycobacterium species.Interactions between β-catenin and transforming growth factor-β signaling pathways mediate epithelial-mesenchymal transition and are dependent on the transcriptional co-activator cAMP-response element-binding protein (CREB)-binding protein (CBP).Esophageal epithelial cells acquire functional characteristics of activated myofibroblasts after undergoing an epithelial to mesenchymal transition.Ligand-independent transforming growth factor-β type I receptor signalling mediates type I collagen-induced epithelial-mesenchymal transition.Intratracheal bleomycin causes airway remodeling and airflow obstruction in mice.Telomerase deficiency does not alter bleomycin-induced fibrosis in mice Integrated analyses identify the involvement of microRNA-26a in epithelial-mesenchymal transition during idiopathic pulmonary fibrosis.DA-Raf-dependent inhibition of the Ras-ERK signaling pathway in type 2 alveolar epithelial cells controls alveolar formation.Alveolar epithelium: beyond the barrier.Chronic respiratory aeroallergen exposure in mice induces epithelial-mesenchymal transition in the large airways Alveolar epithelial cells express mesenchymal proteins in patients with idiopathic pulmonary fibrosis Pathogenesis of idiopathic pulmonary fibrosis Microarray profiling reveals the integrated stress response is activated by halofuginone in mammary epithelial cells.Inhibition of Wnt/beta-catenin/CREB binding protein (CBP) signaling reverses pulmonary fibrosis.A novel, orally active LPA(1) receptor antagonist inhibits lung fibrosis in the mouse bleomycin model.Molecular mechanisms and treatment of radiation-induced lung fibrosis Repetitive intratracheal bleomycin models several features of idiopathic pulmonary fibrosis.Fibrocytes are not an essential source of type I collagen during lung fibrosis.Signaling pathways in the epithelial origins of pulmonary fibrosis Role of the urokinase-fibrinolytic system in epithelial-mesenchymal transition during lung injury.Low-dose paclitaxel ameliorates pulmonary fibrosis by suppressing TGF-β1/Smad3 pathway via miR-140 upregulation.TGFβ signaling in lung epithelium regulates bleomycin-induced alveolar injury and fibroblast recruitment.Endoplasmic reticulum stress enhances fibrotic remodeling in the lungs

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Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

http://www.wikidata.org/entity/Q42021296

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2009年論文

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2009年论文

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name

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

@en

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

@nl

type

label

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

@en

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

@nl

prefLabel

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

@en

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

@nl

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RCCM.200903-0322OC

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American Journal of Respiratory and Critical Care Medicine

P1476

Contribution of epithelial-derived fibroblasts to bleomycin-induced lung fibrosis.

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P2093

Amber L Degryse

http://www.w3.org/2001/XMLSchema#string

Bo Li

http://www.w3.org/2001/XMLSchema#string

David Plieth

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Eric G Neilson

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Harikrishna Tanjore

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Taylor P Sherrill

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Timothy S Blackwell

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Vasiliy V Polosukhin

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Wei Han

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William E Lawson

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P2860

Epithelial-mesenchymal transition and its implications for fibrosis

TGF-beta1 induces human alveolar epithelial to mesenchymal cell transition (EMT)

Epithelial-mesenchymal transitions in tumour progression

Evidence that fibroblasts derive from epithelium during tissue fibrosis

TGF-beta signaling in fibroblasts modulates the oncogenic potential of adjacent epithelia

Alveolar epithelial cell mesenchymal transition develops in vivo during pulmonary fibrosis and is regulated by the extracellular matrix

Conditional abatement of tissue fibrosis using nucleoside analogs to selectively corrupt DNA replication in transgenic fibroblasts.

Myofibroblasts. I. Paracrine cells important in health and disease.

Early restriction of peripheral and proximal cell lineages during formation of the lung.

Respiratory epithelial cell expression of human transforming growth factor-alpha induces lung fibrosis in transgenic mice.

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pulmonary fibrosis

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