Diabetic impairments in NO-mediated endothelial progenitor cell mobilization and homing are reversed by hyperoxia and SDF-1 alpha. - Wikidata - awgldk - TriplyDB

Diabetic impairments in NO-mediated endothelial progenitor cell mobilization and homing are reversed by hyperoxia and SDF-1 alpha.

Diabetic impairments in NO-mediated endothelial progenitor cell mobilization and homing are reversed by hyperoxia and SDF-1 alpha.

http://www.wikidata.org/entity/Q42031332

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Endothelial Progenitor Cells in Diabetic Microvascular Complications: Friends or Foes?Challenges and Opportunities in Drug Delivery for Wound Healing Are Endothelial Progenitor Cells the Real Solution for Cardiovascular Diseases? Focus on Controversies and Perspectives Chemokines in Wound Healing and as Potential Therapeutic Targets for Reducing Cutaneous Scarring Chemokine Regulation of Angiogenesis During Wound Healing The Importance of the CXCL12/CXCR4 Axis in Therapeutic Approaches to Diabetes Mellitus Attenuation Nitric oxide synthases, S-nitrosylation and cardiovascular health: from molecular mechanisms to therapeutic opportunities (review)Endothelial progenitor cell dysfunction in polycystic ovary syndrome: implications for the genesis of cardiovascular diseases Mesenchymal Stem Cells: Rising Concerns over Their Application in Treatment of Type One Diabetes Mellitus Topical insulin accelerates wound healing in diabetes by enhancing the AKT and ERK pathways: a double-blind placebo-controlled clinical trial Remote ischaemic conditioning in the context of type 2 diabetes and neuropathy: the case for repeat application as a novel therapy for lower extremity ulceration Circulating and tissue resident endothelial progenitor cells Notch pathway modulation on bone marrow-derived vascular precursor cells regulates their angiogenic and wound healing potential Antimycotic ciclopirox olamine in the diabetic environment promotes angiogenesis and enhances wound healing Oxidative stress and diabetic complications The potential effect of biological sealants on colorectal anastomosis healing in experimental research involving severe diabetes.Chronic wound repair and healing in older adults: current status and future research.Genetic and epigenetic events in diabetic wound healing.Limb ischemia after iliac ligation in aged mice stimulates angiogenesis without arteriogenesis.Noncontact, low-frequency ultrasound therapy enhances neovascularization and wound healing in diabetic mice.The CXCL12gamma chemokine displays unprecedented structural and functional properties that make it a paradigm of chemoattractant proteins Clinical challenges of chronic wounds: searching for an optimal animal model to recapitulate their complexity Downregulation of ETS rescues diabetes-induced reduction of endothelial progenitor cells.cGMP-dependent protein kinase I is crucial for angiogenesis and postnatal vasculogenesis Comparison of endothelial progenitor cell function in type 2 diabetes with good and poor glycemic control Concise Review: Fat and Furious: Harnessing the Full Potential of Adipose-Derived Stromal Vascular Fraction Recovery from hind limb ischemia is less effective in type 2 than in type 1 diabetic mice: roles of endothelial nitric oxide synthase and endothelial progenitor cells.Substance P accelerates wound healing in type 2 diabetic mice through endothelial progenitor cell mobilization and Yes-associated protein activation CD34+/CD45-dim stem cell mobilization by hyperbaric oxygen - changes with oxygen dosage Effect of vitamin D on endothelial progenitor cells function Identification of E-selectin as a novel target for the regulation of postnatal neovascularization: implications for diabetic wound healing.Lack of CXC chemokine receptor 3 signaling leads to hypertrophic and hypercellular scarring Unacylated ghrelin rescues endothelial progenitor cell function in individuals with type 2 diabetes Local injection of deferoxamine improves neovascularization in ischemic diabetic random flap by increasing HIF-1α and VEGF expression Factors affecting wound healing.Characterization of endothelial progenitor cells mobilization following cutaneous wounding.Heterogeneity in SDF-1 expression defines the vasculogenic potential of adult cardiac progenitor cells.Molecular insights and therapeutic targets for diabetic endothelial dysfunction.Improved culture-based isolation of differentiating endothelial progenitor cells from mouse bone marrow mononuclear cells Enhanced healing of diabetic wounds by topical administration of adipose tissue-derived stromal cells overexpressing stromal-derived factor-1: biodistribution and engraftment analysis by bioluminescent imaging.

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P2860

Diabetic impairments in NO-mediated endothelial progenitor cell mobilization and homing are reversed by hyperoxia and SDF-1 alpha.

http://www.wikidata.org/entity/Q42031332

description

2007 nî lūn-bûn

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2007年の論文

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name

Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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type

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Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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Journal of Clinical Investigation

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Diabetic impairments in NO-med ...... by hyperoxia and SDF-1 alpha.

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April Nedeau

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Donald G Buerk

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Haiying Chen

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Katherine A Gallagher

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Lee J Goldstein

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Min Xiao

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Omaida C Velazquez

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Stephen R Thom

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Zhao-Jun Liu

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P2860

Hyperbaric oxygen therapy for chronic wounds

The neuroblast and angioblast chemotaxic factor SDF-1 (CXCL12) expression is briefly up regulated by reactive astrocytes in brain following neonatal hypoxic-ischemic injury.

Therapeutic stem and progenitor cell transplantation for organ vascularization and regeneration

Isolation of putative progenitor endothelial cells for angiogenesis

HMG-CoA reductase inhibitor mobilizes bone marrow--derived endothelial progenitor cells

Hyperglycemia inhibits endothelial nitric oxide synthase activity by posttranslational modification at the Akt site

Nitric oxide synthase modulates angiogenesis in response to tissue ischemia

Tie-1 and tie-2 define another class of putative receptor tyrosine kinase genes expressed in early embryonic vascular system

Progenitor cell trafficking is regulated by hypoxic gradients through HIF-1 induction of SDF-1

Stimulation of nitric oxide synthase in cerebral cortex due to elevated partial pressures of oxygen: an oxidative stress response.

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1249-1259

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117

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17476357

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