Inhibition of fatty acid amide hydrolase and monoacylglycerol lipase by the anandamide uptake inhibitor VDM11: evidence that VDM11 acts as an FAAH substrate
about
Enzymatic pathways that regulate endocannabinoid signaling in the nervous systemThe endocannabinoid system as a target for the treatment of neurodegenerative disease.Inhibition of monoacylglycerol lipase by troglitazone, N-arachidonoyl dopamine and the irreversible inhibitor JZL184: comparison of two different assays.Anandamide uptake is consistent with rate-limited diffusion and is regulated by the degree of its hydrolysis by fatty acid amide hydrolase.Role of the endogenous cannabinoid system in nicotine addiction: novel insights.Fatty acid-binding proteins transport N-acylethanolamines to nuclear receptors and are targets of endocannabinoid transport inhibitors.The anandamide transport inhibitor AM404 reduces the rewarding effects of nicotine and nicotine-induced dopamine elevations in the nucleus accumbens shell in rats.Inhibition of 2-arachidonoylglycerol catabolism modulates vasoconstriction of rat middle cerebral artery by the thromboxane mimetic, U-46619.The endocannabinoid system: current pharmacological research and therapeutic possibilities.Studies of anandamide accumulation inhibitors in cerebellar granule neurons: comparison to inhibition of fatty acid amide hydrolaseThe cannabinoid system and its pharmacological manipulation--a review, with emphasis upon the uptake and hydrolysis of anandamide.Endocannabinoid- and mGluR5-dependent short-term synaptic depression in an isolated neuron/bouton preparation from the hippocampal CA1 region.The complications of promiscuity: endocannabinoid action and metabolismDivergent effects of anandamide transporter inhibitors with different target selectivity on social play behavior in adolescent rats.No more pain upon Gq-protein-coupled receptor activation: role of endocannabinoids.Cannabinoids and GI Disorders: Endogenous and Exogenous.Acyl-based anandamide uptake inhibitors cause rapid toxicity to C6 glioma cells at pharmacologically relevant concentrations.N-Arachidonoyl Dopamine: A Novel Endocannabinoid and Endovanilloid with Widespread Physiological and Pharmacological Activities.Paradoxical effects of the endocannabinoid uptake inhibitor VDM11 on accumbal neural encoding of reward predictive cues.The Endocannabinoid Reuptake Inhibitor WOBE437 Is Orally Bioavailable and Exerts Indirect Polypharmacological Effects via Different Endocannabinoid Receptors.
P2860
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P2860
Inhibition of fatty acid amide hydrolase and monoacylglycerol lipase by the anandamide uptake inhibitor VDM11: evidence that VDM11 acts as an FAAH substrate
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@en
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@nl
type
label
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@en
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@nl
prefLabel
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@en
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@nl
P2860
P921
P356
P1476
Inhibition of fatty acid amide ...... DM11 acts as an FAAH substrate
@en
P2093
Christopher J Fowler
Séverine Vandevoorde
P2860
P304
P356
10.1038/SJ.BJP.0706253
P407
P577
2005-08-01T00:00:00Z