Cell-Type Specific Channelopathies in the Prefrontal Cortex of the fmr1-/y Mouse Model of Fragile X Syndrome.
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mTOR referees memory and disease through mRNA repression and competition.Increased Persistent Sodium Current Causes Neuronal Hyperexcitability in the Entorhinal Cortex of Fmr1 Knockout Mice.MicroRNA-Mediated Downregulation of the Potassium Channel Kv4.2 Contributes to Seizure Onset.Excitability is increased in hippocampal CA1 pyramidal cells of Fmr1 knockout mice.Identifying specific prefrontal neurons that contribute to autism-associated abnormalities in physiology and social behavior.Increased transient Na+ conductance and action potential output in layer 2/3 prefrontal cortex neurons of the fmr1-/y mouse.Systems-based analysis of dendritic nonlinearities reveals temporal feature extraction in mouse L5 cortical neurons.Rescue of hyperexcitability in hippocampal CA1 neurons from Mecp2 (-/y) mouse through surface potential neutralization.Regenerative glutamate release in the hippocampus of Rett syndrome model mice
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P2860
Cell-Type Specific Channelopathies in the Prefrontal Cortex of the fmr1-/y Mouse Model of Fragile X Syndrome.
description
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Cell-Type Specific Channelopat ...... e Model of Fragile X Syndrome.
@en
Cell-Type Specific Channelopat ...... e Model of Fragile X Syndrome.
@nl
type
label
Cell-Type Specific Channelopat ...... e Model of Fragile X Syndrome.
@en
Cell-Type Specific Channelopat ...... e Model of Fragile X Syndrome.
@nl
prefLabel
Cell-Type Specific Channelopat ...... e Model of Fragile X Syndrome.
@en
Cell-Type Specific Channelopat ...... e Model of Fragile X Syndrome.
@nl
P2860
P1433
P1476
Cell-Type Specific Channelopat ...... se Model of Fragile X Syndrome
@en
P2093
Brian E Kalmbach
Daniel Johnston
P2860
P356
10.1523/ENEURO.0114-15.2015
P577
2015-11-17T00:00:00Z