The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
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Molecular crosstalk between apoptosis, necroptosis, and survival signalingPoly-ubiquitination in TNFR1-mediated necroptosisUbiquitin modificationsUbiquitylation as a Rheostat for TCR Signaling: From Targeted Approaches Toward Global ProfilingRegulation of Met1-linked polyubiquitin signalling by the deubiquitinase OTULINDeubiquitinating enzymes in cellular signaling and disease regulationDeconstructing innate immune signaling in myelodysplastic syndromesNF-κB in inflammation and renal diseasesLinear ubiquitination in immunityTCR signaling to NF-κB and mTORC1: Expanding roles of the CARMA1 complexThe paracaspase MALT1 mediates CARD14-induced signaling in keratinocytesEpithelial Cell Regulation of Allergic Diseases.USP18 lack in microglia causes destructive interferonopathy of the mouse brain.Vitamin E γ-Tocotrienol Inhibits Cytokine-Stimulated NF-κB Activation by Induction of Anti-Inflammatory A20 via Stress Adaptive Response Due to Modulation of Sphingolipids.Ubiquitin-specific Protease 20 Regulates the Reciprocal Functions of β-Arrestin2 in Toll-like Receptor 4-promoted Nuclear Factor κB (NFκB) Activation.RIPK3 deficiency or catalytically inactive RIPK1 provides greater benefit than MLKL deficiency in mouse models of inflammation and tissue injury.DUBs, the regulation of cell identity and disease.Cystic Fibrosis from Laboratory to Bedside: The Role of A20 in NF-κB-Mediated Inflammation.Topology, dynamics, and heterogeneity in immune signaling.Formation and removal of poly-ubiquitin chains in the regulation of tumor necrosis factor-induced gene activation and cell death.More to Life than NF-κB in TNFR1 SignalingCYLD, A20 and OTULIN deubiquitinases in NF-κB signaling and cell death: so similar, yet so different.The role of hybrid ubiquitin chains in the MyD88 and other innate immune signalling pathways.Limiting inflammation-the negative regulation of NF-κB and the NLRP3 inflammasome.The ubiquitin-specific protease USP8 is critical for the development and homeostasis of T cells.A20 suppresses canonical Smad-dependent fibroblast activation: novel function for an endogenous inflammatory modulator.Replication defective viral genomes exploit a cellular pro-survival mechanism to establish paramyxovirus persistence.A20: attractive without showing cleavage.A20 prevents obesity-induced development of cardiac dysfunction.Deubiquitinases A20 and CYLD modulate costimulatory signaling via CD137 (4-1BB).Nuclear Factor-kappaB in Autoimmunity: Man and Mouse.A20 regulates canonical wnt-signaling through an interaction with RIPK4.Ubiquitination regulation of inflammatory responses through NF-κB pathway.TNIP1 in Autoimmune Diseases: Regulation of Toll-like Receptor Signaling
P2860
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P2860
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
description
2014 nî lūn-bûn
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2014年の論文
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2014年学术文章
@wuu
2014年学术文章
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2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
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2014年學術文章
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2014年學術文章
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name
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
@en
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
@nl
type
label
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
@en
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
@nl
prefLabel
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
@en
The deubiquitinase activity of A20 is dispensable for NF-κB signaling.
@nl
P2093
P2860
P356
P1433
P1476
The deubiquitinase activity of A20 is dispensable for NF-κB signaling
@en
P2093
Chozha Vendan Rathinam
Sankar Ghosh
P2860
P304
P356
10.15252/EMBR.201338305
P577
2014-05-30T00:00:00Z