Acute alpha 1-adrenergic stimulation of cardiac protein synthesis may involve increased intracellular pH and protein kinase activity.
about
Protein kinase C phosphorylation of cardiac troponin T decreases Ca(2+)-dependent actomyosin MgATPase activity and troponin T binding to tropomyosin-F-actin complexbeta-Adrenergic agonists increase phosphorylation of elongation factor 2 in cardiomyocytes without eliciting calcium-independent eEF2 kinase activity.MAPK signaling pathways are needed for survival of H9c2 cardiac myoblasts under extracellular alkalosis.Correlations between cardiac protein synthesis rates, intracellular pH and the concentrations of creatine metabolites.The effects of the exogenous provision of lactate and the endogenous production of lactate on protein synthesis in the heart.Hypertrophic responsiveness of cardiomyocytes to alpha- or beta-adrenoceptor stimulation requires sodium-proton-exchanger-1 (NHE-1) activation but not cellular alkalization.
P2860
Acute alpha 1-adrenergic stimulation of cardiac protein synthesis may involve increased intracellular pH and protein kinase activity.
description
1991 nî lūn-bûn
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1991年の論文
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1991年学术文章
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1991年学术文章
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1991年学术文章
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1991年学术文章
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1991年學術文章
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1991年學術文章
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name
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@en
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@nl
type
label
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@en
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@nl
prefLabel
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@en
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@nl
P2093
P2860
P356
P1433
P1476
Acute alpha 1-adrenergic stimu ...... H and protein kinase activity.
@en
P2093
Gaitanaki CJ
Hatchett RJ 4th
P2860
P304
P356
10.1042/BJ2730347
P407
P478
P577
1991-01-01T00:00:00Z