p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2. - Wikidata - awgldk - TriplyDB

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

http://www.wikidata.org/entity/Q42090504

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UVB irradiation regulates ERK1/2- and p53-dependent thrombomodulin expression in human keratinocytes Assessing the carcinogenic potential of low-dose exposures to chemical mixtures in the environment: focus on the cancer hallmark of tumor angiogenesis Minnelide/Triptolide Impairs Mitochondrial Function by Regulating SIRT3 in P53-Dependent Manner in Non-Small Cell Lung Cancer Simvastatin-induced sphingosine 1-phosphate receptor 1 expression is KLF2-dependent in human lung endothelial cells.Cytotoxic effects of docetaxel as a candidate drug of drug-eluting stent on human umbilical vein endothelial cells and the signaling pathway of cell migration inhibition, adhesion delay and shape change Global effect of inauhzin on human p53-responsive transcriptome Disturbed flow: p53 SUMOylation in the turnover of endothelial cells The anti-hypercholesterolemic effect of low p53 expression protects vascular endothelial function in mice Epigenetic upregulation of p66shc mediates low-density lipoprotein cholesterol-induced endothelial cell dysfunction Reactive Oxygen Species, SUMOylation, and Endothelial Inflammation Phosphorylation of protein inhibitor of activated STAT1 (PIAS1) by MAPK-activated protein kinase-2 inhibits endothelial inflammation via increasing both PIAS1 transrepression and SUMO E3 ligase activity.Efficacy of flavonoids in the management of high blood pressure.Lipopolysaccharide Downregulates Kruppel-Like Factor 2 (KLF2) via Inducing DNMT1-Mediated Hypermethylation in Endothelial Cells.Disturbed Flow-Induced Endothelial Proatherogenic Signaling Via Regulating Post-Translational Modifications and Epigenetic Events.Chemotherapy-mediated p53-dependent DNA damage response in clear cell renal cell carcinoma: role of the mTORC1/2 and hypoxia-inducible factor pathways Sub-cellular localization specific SUMOylation in the heart.Mitochondrial Ca2+ transport in the endothelium: regulation by ions, redox signalling and mechanical forces.Allopurinol protects human glomerular endothelial cells from high glucose-induced reactive oxygen species generation, p53 overexpression and endothelial dysfunction.The Redox-sensitive Induction of the Local Angiotensin System Promotes Both Premature and Replicative Endothelial Senescence: Preventive Effect of a Standardized Crataegus Extract.Canonical Wnt signaling induces vascular endothelial dysfunction via p66Shc-regulated reactive oxygen species.Krüppel-Like Factors in Vascular Inflammation: Mechanistic Insights and Therapeutic Potential.Vascular senescence and ageing: a role for the MEOX proteins in promoting endothelial dysfunction.

P2860

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P2860

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

http://www.wikidata.org/entity/Q42090504

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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2010年论文

@zh

2010年论文

@zh-cn

name

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@en

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@nl

type

label

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@en

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@nl

prefLabel

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@en

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@nl

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P356

ATVBAHA.110.215061

P1433

Arteriosclerosis, Thrombosis, and Vascular Biology

P1476

p53 impairs endothelial function by transcriptionally repressing Kruppel-Like Factor 2.

@en

P2093

Ajay Kumar

http://www.w3.org/2001/XMLSchema#string

Asma Naqvi

http://www.w3.org/2001/XMLSchema#string

Cuk-Seong Kim

http://www.w3.org/2001/XMLSchema#string

Jeremy Dericco

http://www.w3.org/2001/XMLSchema#string

Kaikobad Irani

http://www.w3.org/2001/XMLSchema#string

Mukesh K Jain

http://www.w3.org/2001/XMLSchema#string

Saet-Byel Jung

http://www.w3.org/2001/XMLSchema#string

Timothy A Hoffman

http://www.w3.org/2001/XMLSchema#string

Zhiyong Lin

http://www.w3.org/2001/XMLSchema#string

P2860

A novel mechanism for p53 to regulate its target gene ECK in signaling apoptosis

Isolation, genomic structure, and expression of human erythroid Krüppel-like factor (EKLF)

Histone deacetylases (HDACs): characterization of the classical HDAC family

p53, the cellular gatekeeper for growth and division

P66shc regulates endothelial NO production and endothelium-dependent vasorelaxation: implications for age-associated vascular dysfunction

Transcriptional repression of Kruppel like factor-2 by the adaptor protein p66shc.

Transforming growth factor-beta 1 inhibition of macrophage activation is mediated via Smad3.

p53-Dependent transcriptional repression of c-myc is required for G1 cell cycle arrest.

Integration of flow-dependent endothelial phenotypes by Kruppel-like factor 2

p300/CBP/p53 interaction and regulation of the p53 response.

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133-141

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scholarly article

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10.1161/ATVBAHA.110.215061

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1

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31

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2010-10-14T00:00:00Z

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47429392

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20947822

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3064482

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