Cdc25A phosphatase: a key cell cycle protein that regulates neuron death in disease and development.

about

Cdc25A phosphatase: a key cell cycle protein that regulates neuron death in disease and development.

Item

http://www.wikidata.org/entity/Q42093273

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Cdc25A phosphatase: a key cell ...... th in disease and development.

@en

Cdc25A phosphatase: a key cell ...... th in disease and development.

@nl

type

Item

label

Cdc25A phosphatase: a key cell ...... th in disease and development.

@en

Cdc25A phosphatase: a key cell ...... th in disease and development.

@nl

prefLabel

Cdc25A phosphatase: a key cell ...... th in disease and development.

@en

Cdc25A phosphatase: a key cell ...... th in disease and development.

@nl

P1476

Cdc25A phosphatase: a key cell ...... ath in disease and development

@en

P2093

Lloyd A Greene

http://www.w3.org/2001/XMLSchema#string

Nandini Chatterjee

http://www.w3.org/2001/XMLSchema#string

Priyankar Sanphui

http://www.w3.org/2001/XMLSchema#string

Subhas Chandra Biswas

http://www.w3.org/2001/XMLSchema#string

P2860

Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

FoxO3a is activated and executes neuron death via Bim in response to β-amyloid

Cell cycle control by the CDC25 phosphatases

Role and regulation of Cdc25A phosphatase in neuron death induced by NGF deprivation or β-amyloid

Cell cycle molecules define a pathway required for neuron death in development and disease.

The cell cycle Cdc25A tyrosine phosphatase is activated in degenerating postmitotic neurons in Alzheimer's disease.

The insulin/Akt signaling pathway is targeted by intracellular beta-amyloid

Bim is a direct target of a neuronal E2F-dependent apoptotic pathway.

The Chk1/Cdc25A pathway as activators of the cell cycle in neuronal death induced by camptothecin.

A feed-forward loop involving Trib3, Akt and FoxO mediates death of NGF-deprived neurons

P304

e2692

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/CDDIS.2017.115

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Stav Kemeny

P577

2017-03-23T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28333146

http://www.w3.org/2001/XMLSchema#string

P932

5386521

http://www.w3.org/2001/XMLSchema#string