CRISPR-Cas9 genome editing of a single regulatory element nearly abolishes target gene expression in mice--brief report.
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Making sense of GWAS: using epigenomics and genome engineering to understand the functional relevance of SNPs in non-coding regions of the human genomeThe short and long of noncoding sequences in the control of vascular cell phenotypesLoss of LMOD1 impairs smooth muscle cytocontractility and causes megacystis microcolon intestinal hypoperistalsis syndrome in humans and mice.Culture time of vitrified/warmed zygotes before microinjection affects the production efficiency of CRISPR-Cas9-mediated knock-in mice.CRISPR/Cas9 targeting events cause complex deletions and insertions at 17 sites in the mouse genome.Disruption of the Axonal Trafficking of Tyrosine Hydroxylase mRNA Impairs Catecholamine Biosynthesis in the Axons of Sympathetic NeuronsProduction of knockout mice by DNA microinjection of various CRISPR/Cas9 vectors into freeze-thawed fertilized oocytesSerum response factor regulates smooth muscle contractility via myotonic dystrophy protein kinases and L-type calcium channelsA CRISPR Path to Engineering New Genetic Mouse Models for Cardiovascular ResearchIntegrative functional genomics identifies regulatory mechanisms at coronary artery disease loci.Ultra-superovulation for the CRISPR-Cas9-mediated production of gene-knockout, single-amino-acid-substituted, and floxed mice.Genome-wide compendium and functional assessment of in vivo heart enhancers.Noncoding RNAs in smooth muscle cell homeostasis: implications in phenotypic switch and vascular disorders.In vivo and in vitro disease modeling with CRISPR/Cas9.A history of genome editing in mammals.Genome Editing: The Recent History and Perspective in Cardiovascular Diseases.Novel Thrombotic Function of a Human SNP in STXBP5 Revealed by CRISPR/Cas9 Gene Editing in Mice.In vivo genome editing thrives with diversified CRISPR technologies.Nexilin/NEXN controls actin polymerization in smooth muscle and is regulated by myocardin family coactivators and YAP
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P2860
CRISPR-Cas9 genome editing of a single regulatory element nearly abolishes target gene expression in mice--brief report.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
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2014年论文
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2014年论文
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name
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@en
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@nl
type
label
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@en
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@nl
prefLabel
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@en
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@nl
P2093
P2860
P1476
CRISPR-Cas9 genome editing of ...... ression in mice--brief report.
@en
P2093
Albert W Cheng
Christine K Christie
Joseph M Miano
Orazio J Slivano
P2860
P304
P356
10.1161/ATVBAHA.114.305017
P407
P577
2014-12-23T00:00:00Z