Inhibition of NOX2 reduces locomotor impairment, inflammation, and oxidative stress after spinal cord injury.

Inhibition of NOX2 reduces locomotor impairment, inflammation, and oxidative stress after spinal cord injury.

Item
http://www.wikidata.org/entity/Q42125632
Therapeutic effect of apocynin through antioxidant activity and suppression of apoptosis and inflammation after spinal cord injury.Anti-Inflammatory Small Molecules To Treat Seizures and Epilepsy: From Bench to BedsideNOX2 deficiency alters macrophage phenotype through an IL-10/STAT3 dependent mechanism: implications for traumatic brain injuryThe Polarization States of Microglia in TBI: A New Paradigm for Pharmacological Intervention.Central Nervous System Injury and Nicotinamide Adenine Dinucleotide Phosphate Oxidase: Oxidative Stress and Therapeutic Targets.NADPH oxidase isoform expression is temporally regulated and may contribute to microglial/macrophage polarization after spinal cord injury.Nanofiber Scaffolds as Drug Delivery Systems to Bridge Spinal Cord Injury.NOX2 drives M1-like microglial/macrophage activation and neurodegeneration following experimental traumatic brain injury.Activation of p47phox as a Mechanism of Bupivacaine-Induced Burst Production of Reactive Oxygen Species and Neural Toxicity.Age exacerbates microglial activation, oxidative stress, inflammatory and NOX2 gene expression, and delays functional recovery in a middle-aged rodent model of spinal cord injury.Deficiency in the voltage-gated proton channel Hv1 increases M2 polarization of microglia and attenuates brain damage from photothrombotic ischemic stroke.Targeting the NLRP3 inflammasome to attenuate spinal cord injury in mice.Age increases reactive oxygen species production in macrophages and potentiates oxidative damage after spinal cord injury.Dl-3-n-butylphthalide prevents the disruption of blood-spinal cord barrier via inhibiting endoplasmic reticulum stress following spinal cord injury.Neuroprotective mechanisms of rutin for spinal cord injury through anti-oxidation and anti-inflammation and inhibition of p38 mitogen activated protein kinase pathway.Nanoparticle-Delivered IRF5 siRNA Facilitates M1 to M2 Transition, Reduces Demyelination and Neurofilament Loss, and Promotes Functional Recovery After Spinal Cord Injury in Mice.A Single Dose of Atorvastatin Applied Acutely after Spinal Cord Injury Suppresses Inflammation, Apoptosis, and Promotes Axon Outgrowth, Which Might Be Essential for Favorable Functional Outcome.
P2860

Inhibition of NOX2 reduces locomotor impairment, inflammation, and oxidative stress after spinal cord injury.

Item
http://www.wikidata.org/entity/Q42125632
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@en
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@nl
type
label
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@en
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@nl
prefLabel
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@en
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P478
P50
P577
P5875
P6179
P698
P932
P356
P1433
P1476
Inhibition of NOX2 reduces loc ...... ress after spinal cord injury.
@en
P2093
P2860
P2888
P304
P31
P356
P478
P50
P577
P5875
P6179
P698
P932