Loss of muscarinic M1 receptor exacerbates Alzheimer's disease-like pathology and cognitive decline.
about
IGF2 ameliorates amyloidosis, increases cholinergic marker expression and raises BMP9 and neurotrophin levels in the hippocampus of the APPswePS1dE9 Alzheimer's disease model mice.Exome sequencing of case-unaffected-parents trios reveals recessive and de novo genetic variants in sporadic ALS.Neurotransmitter receptors and cognitive dysfunction in Alzheimer's disease and Parkinson's disease.Preface.Mifepristone alters amyloid precursor protein processing to preclude amyloid beta and also reduces tau pathology.Aspirin-triggered lipoxin A4 stimulates alternative activation of microglia and reduces Alzheimer disease-like pathology in mice.Why an M1 Antagonist Could Be a More Selective Model for Memory Impairment than Scopolamine.BMP9 ameliorates amyloidosis and the cholinergic defect in a mouse model of Alzheimer's disease.Stress hormone leads to memory deficits and altered tau phosphorylation in a model of Alzheimer's disease.Cholinergic modulation of amyloid precursor protein processing with emphasis on M1 muscarinic receptor: perspectives and challenges in treatment of Alzheimer's disease.Muscarinic acetylcholine receptors: novel opportunities for drug development.Insights into the structural biology of G-protein coupled receptors impacts drug design for central nervous system neurodegenerative processes.Interaction of basal forebrain cholinergic neurons with the glucocorticoid system in stress regulation and cognitive impairment.Cholinergic System and Post-translational Modifications: An Insight on the Role in Alzheimer's DiseaseNoradrenergic sympathetic sprouting and cholinergic reinnervation maintains non-amyloidogenic processing of AβPP.Muscarinic acetylcholine receptors act in synergy to facilitate learning and memory.Physiological roles of CNS muscarinic receptors gained from knockout mice.Differentially disrupted functional connectivity of the subregions of the inferior parietal lobule in Alzheimer's disease.Cognitive Decline in Preclinical Alzheimer's Disease: Amyloid-Beta versus Tauopathy.Acute Effects of Muscarinic M1 Receptor Modulation on AβPP Metabolism and Amyloid-β Levels in vivo: A Microdialysis Study.Direct excitation of parvalbumin-positive interneurons by M1 muscarinic acetylcholine receptors: roles in cellular excitability, inhibitory transmission and cognition.Therapeutics of Neurotransmitters in Alzheimer's Disease.Effects of sub-chronic donepezil on brain Abeta and cognition in a mouse model of Alzheimer's disease.Prediction of consensus binding mode geometries for related chemical series of positive allosteric modulators of adenosine and muscarinic acetylcholine receptors.Therapy of Dredging the Bowels Enhanced the Neuroprotective Effect of Nourishing Kidney Herbs on Hippocampal Cholinergic System in Alzheimer's Disease Model Rat Induced by A 1-42
P2860
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P2860
Loss of muscarinic M1 receptor exacerbates Alzheimer's disease-like pathology and cognitive decline.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@en
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@nl
type
label
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@en
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@nl
prefLabel
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@en
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@nl
P2093
P2860
P1476
Loss of muscarinic M1 receptor ...... thology and cognitive decline.
@en
P2093
Antonella Caccamo
Avraham Fisher
David H Cribbs
Frank M LaFerla
Masashi Kitazawa
Tatiana Estrada-Hernandez
P2860
P304
P356
10.1016/J.AJPATH.2011.04.041
P407
P577
2011-06-23T00:00:00Z