The Campylobacter jejuni response regulator, CbrR, modulates sodium deoxycholate resistance and chicken colonization.
about
Regulation of oxidative stress resistance in Campylobacter jejuni, a microaerophilic foodborne pathogenTranscriptional regulation of the CmeABC multidrug efflux pump and the KatA catalase by CosR in Campylobacter jejuni.CmeR functions as a pleiotropic regulator and is required for optimal colonization of Campylobacter jejuni in vivo.A PAS domain-containing regulator controls flagella-flagella interactions in Campylobacter jejuniEnvironmental survival mechanisms of the foodborne pathogen Campylobacter jejuni.High-throughput sequencing of Campylobacter jejuni insertion mutant libraries reveals mapA as a fitness factor for chicken colonization.RNAseq Reveals Complex Response of Campylobacter jejuni to Ovine Bile and In vivo Gallbladder Environment.Campylobacter jejuni dsb gene expression is regulated by iron in a Fur-dependent manner and by a translational coupling mechanismRegulation of oxidative stress response by CosR, an essential response regulator in Campylobacter jejuni.Amino-terminal residues dictate the export efficiency of the Campylobacter jejuni filament proteins via the flagellum.Bile salts modulate expression of the CmeABC multidrug efflux pump in Campylobacter jejuniCampylobacter jejuni CsrA complements an Escherichia coli csrA mutation for the regulation of biofilm formation, motility and cellular morphology but not glycogen accumulationUse of a rabbit soft tissue chamber model to investigate campylobacter jejuni-host interactions.Divergent distribution of the sensor kinase CosS in non-thermotolerant campylobacter species and its functional incompatibility with the response regulator CosR of Campylobacter jejuniCapA, an autotransporter protein of Campylobacter jejuni, mediates association with human epithelial cells and colonization of the chicken gutCharacterization of genetically matched isolates of Campylobacter jejuni reveals that mutations in genes involved in flagellar biosynthesis alter the organism's virulence potential.Characterization of a novel bile-inducible operon encoding a two-component regulatory system in Lactobacillus acidophilus.Cj1136 is required for lipooligosaccharide biosynthesis, hyperinvasion, and chick colonization by Campylobacter jejuni.The Campylobacter jejuni CiaC virulence protein is secreted from the flagellum and delivered to the cytosol of host cells.High-Frequency Variation of Purine Biosynthesis Genes Is a Mechanism of Success in Campylobacter jejuni.The acid adaptive tolerance response in Campylobacter jejuni induces a global response, as suggested by proteomics and microarrays.Hyperosmotic stress response of Campylobacter jejuni.Dual Repression of the Multidrug Efflux Pump CmeABC by CosR and CmeR in Campylobacter jejuni.Transcriptional Profile during Deoxycholate-Induced Sporulation in a Clostridium perfringens Isolate Causing Foodborne Illness.Advances in Campylobacter biology and implications for biotechnological applications.The CprS sensor kinase of the zoonotic pathogen Campylobacter jejuni influences biofilm formation and is required for optimal chick colonization.Colonization factors of Campylobacter jejuni in the chicken gutThe role of probiotics in the inhibition of Campylobacter jejuni colonization and virulence attenuation.Survival of the Fittest: How Bacterial Pathogens Utilize Bile To Enhance Infection.Regulation of antimicrobial resistance by extracytoplasmic function (ECF) sigma factors.Host cell contact induces expression of virulence factors and VieA, a cyclic di-GMP phosphodiesterase, in Vibrio choleraeIdentification of Campylobacter jejuni genes involved in its interaction with epithelial cells.Culture of Campylobacter jejuni with sodium deoxycholate induces virulence gene expression.Role of alkyl hydroperoxide reductase (AhpC) in the biofilm formation of Campylobacter jejuni.FdhTU-modulated formate dehydrogenase expression and electron donor availability enhance recovery of Campylobacter jejuni following host cell infection.Characterization of Campylobacter jejuni RacRS reveals roles in the heat shock response, motility, and maintenance of cell length homogeneity.Identification of genetic loci that contribute to Campylobacter resistance to fowlicidin-1, a chicken host defense peptideThe Campylobacter jejuni PhosS/PhosR operon represents a non-classical phosphate-sensitive two-component system.The food-borne pathogen Campylobacter jejuni responds to the bile salt deoxycholate with countermeasures to reactive oxygen species.The bile salt sodium taurocholate induces Campylobacter jejuni outer membrane vesicle production and increases OMV-associated proteolytic activity.
P2860
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P2860
The Campylobacter jejuni response regulator, CbrR, modulates sodium deoxycholate resistance and chicken colonization.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
The Campylobacter jejuni respo ...... ance and chicken colonization.
@en
The Campylobacter jejuni respo ...... ance and chicken colonization.
@nl
type
label
The Campylobacter jejuni respo ...... ance and chicken colonization.
@en
The Campylobacter jejuni respo ...... ance and chicken colonization.
@nl
prefLabel
The Campylobacter jejuni respo ...... ance and chicken colonization.
@en
The Campylobacter jejuni respo ...... ance and chicken colonization.
@nl
P2093
P2860
P1476
The Campylobacter jejuni respo ...... ance and chicken colonization.
@en
P2093
Brian H Raphael
Gary A Flom
Julian M Ketley
Michael E Konkel
Sonia Pereira
P2860
P304
P356
10.1128/JB.187.11.3662-3670.2005
P50
P577
2005-06-01T00:00:00Z