Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis - Wikidata - awgldk - TriplyDB

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

http://www.wikidata.org/entity/Q42138750

about

Noncoding RNAs, Emerging Regulators of Skeletal Muscle Development and Diseases Systemic Delivery of scAAV8-Encoded MiR-29a Ameliorates Hepatic Fibrosis in Carbon Tetrachloride-Treated Mice LncRNA Dum interacts with Dnmts to regulate Dppa2 expression during myogenic differentiation and muscle regeneration Combined isosorbide dinitrate and ibuprofen as a novel therapy for muscular dystrophies: evidence from Phase I studies in healthy volunteers.MicroRNA-29 overexpression by adeno-associated virus suppresses fibrosis and restores muscle function in combination with micro-dystrophin MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms Genetic polymorphism and chronic obstructive pulmonary disease.Mega roles of microRNAs in regulation of skeletal muscle health and disease Mast cell plasticity and sphingosine-1-phosphate in immunity, inflammation and cancer.Loss of miR-29b following acute ischemic stroke contributes to neural cell death and infarct size.Satellite cells from dystrophic muscle retain regenerative capacity.Do epigenetic events take place in the vastus lateralis of patients with mild chronic obstructive pulmonary disease?Disruption of miR-29 Leads to Aberrant Differentiation of Smooth Muscle Cells Selectively Associated with Distal Lung Vasculature.Pathophysiological role of microRNA-29 in pancreatic cancer stroma Genome-wide Mechanosensitive MicroRNA (MechanomiR) Screen Uncovers Dysregulation of Their Regulatory Networks in the mdm Mouse Model of Muscular Dystrophy.Multi-level omics analysis in a murine model of dystrophin loss and therapeutic restoration.miR-29 targets Akt3 to reduce proliferation and facilitate differentiation of myoblasts in skeletal muscle development.Molecular mechanism of sphingosine-1-phosphate action in Duchenne muscular dystrophy.Malat1 regulates myogenic differentiation and muscle regeneration through modulating MyoD transcriptional activity MicroRNA-29 in the adaptive immune system: setting the threshold.Ongoing therapeutic trials and outcome measures for Duchenne muscular dystrophy.Therapeutic targeting of non-coding RNAs.MicroRNA in skeletal muscle development, growth, atrophy, and disease.Integration of miRNA and mRNA expression profiles reveals microRNA-regulated networks during muscle wasting in cardiac cachexia.The implications on clinical diagnostics of using microRNA-based biomarkers in exercise.Linc-YY1 promotes myogenic differentiation and muscle regeneration through an interaction with the transcription factor YY1.LncFunNet: an integrated computational framework for identification of functional long noncoding RNAs in mouse skeletal muscle cells.The role of MicroRNAs in COPD muscle dysfunction and mass loss: implications on the clinic.Noncoding RNAs in the regulation of skeletal muscle biology in health and disease.Genome-wide survey by ChIP-seq reveals YY1 regulation of lincRNAs in skeletal myogenesis.Epigenetic regulation of muscle development.A novel target of microRNA-29, Ring1 and YY1-binding protein (Rybp), negatively regulates skeletal myogenesis A novel miR-193a-5p-YY1-APC regulatory axis in human endometrioid endometrial adenocarcinoma.Laminin α2 Chain-Deficiency is Associated with microRNA Deregulation in Skeletal Muscle and Plasma.Restoration of muscle strength in dystrophic muscle by angiotensin-1-7 through inhibition of TGF-β signalling.Cardiac myocyte miR-29 promotes pathological remodeling of the heart by activating Wnt signaling.Osteoglycin inhibition by microRNA miR-155 impairs myogenesis.MicroRNA expression patterns in canine mammary cancer show significant differences between metastatic and non-metastatic tumours.Noncoding RNAs and Duchenne muscular dystrophy.Muscle-specific microRNA-206 targets multiple components in dystrophic skeletal muscle representing beneficial adaptations.

P2860

Q26799919-67086555-5DA9-4F0E-ABC7-1E65EBF819AB Q28546811-AD216F46-1972-4A8B-852A-BBA6D44C6954 Q28591298-EF374680-5AE2-4A6F-858F-06CA28930C7C Q33599468-6D84E53B-40C2-4E24-B726-CCD12A43BC05 Q33631076-4E385CD9-6485-459B-BA95-6EF8F6593933 Q33685967-88103455-EC71-455D-83F3-BFC553C38B06 Q33704044-D25E861F-236B-4781-8F1E-31E32768E8C3 Q33810713-238C48F4-0037-47F4-8AAD-7DE7F11BBE34 Q34482185-B13E97A0-C8C5-43F6-90BE-7757AF310235 Q34697381-D405966A-6588-444A-ADD0-A3421FA62658 Q35009253-7822E475-6082-4E0C-A91C-F42A13215C3B Q35205327-9980291B-6A88-41FA-8624-66B5A1EAC010 Q35644474-B77185C2-E699-4C60-BA8D-E74288F38250 Q35765000-841D4BA5-70BD-4431-AEC8-82F11AD2E365 Q36139893-5AAED93C-460F-4071-93AA-4BD0615A39E4 Q36252005-5DAE2992-3556-46B4-80CC-F473D9D5AA3C Q36973754-59DE7D22-5100-4AAB-A684-3B5AA7A3DBD2 Q37438488-8599E560-A8CE-4C44-9E24-48695A3BF928 Q37697734-558FAF13-8CA0-4B51-A256-EE39534EC10C Q38042805-2A3811D5-8D9A-4750-9296-1F985E0BFF4E Q38115148-CF262F98-881C-476E-B51C-ADF4C6F5662E Q38119545-831BF01D-FA34-40FE-A28F-9FD024A1B993 Q38212617-0BF22941-92EF-48D6-8C0F-5C5B88446B7B Q38430443-79A17820-27E6-4CF0-ADFC-25BA14706339 Q38480828-983AF82C-C112-4E3A-B322-F88ED1C644CC Q38812278-E6A7FB39-37E6-4985-A448-A607877C0B6C Q38853026-D0ED6D7E-918C-4FAE-B39B-982F10CFA37C Q38878269-0C2AE5D9-1A3A-41ED-8B4B-692AD365C053 Q38885730-4D2D7B98-D08D-4518-B8CF-8A249DF2521C Q39110624-CE2DD65D-740B-4645-B27B-A43A94140660 Q39206248-8C750714-EACC-4606-B447-4432D365DA43 Q39337764-1BD06608-0C3B-4EDB-A740-4091B1111B12 Q39566547-DE91AA60-1B2B-4201-AD13-BD1EDF099C58 Q42719076-BA5E3476-BE8D-473A-9EEB-1E516517F935 Q44817032-AF4168BC-F27E-4F33-B8E0-800F47611D18 Q46632595-CE1038A6-A033-4464-B91C-63D940472266 Q47157200-4C3E900A-28FD-444F-BC7D-8BA70377B329 Q47633999-D4E5182F-83F4-489E-BA05-B3AC2B4F0AC3 Q50479884-B020D9DC-23AC-46E6-A5F9-C7B01510AF41 Q51243987-D5FA8DED-2FF0-4E40-9DDC-1D2FB03BAB02

P2860

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

http://www.wikidata.org/entity/Q42138750

description

2012 nî lūn-bûn

@nan

2012年の論文

@ja

2012年論文

@yue

2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

@wuu

2012年论文

@zh

2012年论文

@zh-cn

name

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@en

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@nl

type

label

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@en

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@nl

prefLabel

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@en

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@nl

P1433

Q42138750-7B467344-94B3-4F9C-9177-C90AF9E77EEE

P1476

Q42138750-3C3745D6-E922-40F1-9293-C4559E3EE4A4

P2093

Q42138750-0F206937-E923-473D-BD32-FB57F5386532

Q42138750-29364EBE-1532-4D97-9255-203411CDBA9A

Q42138750-487FC45B-C6FF-4FCF-9CDB-7B72C5E185BA

Q42138750-49E30675-CE67-431B-87F8-3EDF27DB42C4

Q42138750-5A30537A-BB49-4757-8F1B-2EE3AD9C0D2F

Q42138750-F4F00292-5EE1-4FB7-9BAA-D03A2582700C

P2860

Q42138750-042A7638-E8E1-4B57-8D99-AD48321AA6A9

Q42138750-095E80B1-302F-4C62-9B18-FDF2CED07A1C

Q42138750-0BA51CC6-961F-4E3C-BFA7-7799D2307884

Q42138750-0C6AE2F7-53E1-40BC-8DD6-8C416E71889B

Q42138750-1190F90E-D81C-4391-98FC-AB1D5ECC58E4

Q42138750-1395FE10-B29F-4038-8717-EC4F6481988E

Q42138750-1572215D-62CB-4301-9A40-B7F6CFC6431F

Q42138750-1C9B65CD-C96D-4D65-99D6-EAEBBCB7F797

Q42138750-223D144A-A203-41CA-90CF-B85FFD7CA644

Q42138750-2B0BF5A1-E340-4B79-9E3C-460DA53A04AF

P304

Q42138750-0061FDBE-3ADE-44D3-93D2-2C062DC105AB

P31

Q42138750-1AD726B2-943B-45D2-BC5D-8B71835FF92E

P356

Q42138750-FF298938-4F1B-4C4D-95EE-81D5E217FC06

P433

Q42138750-5F480824-B147-4087-B14D-59BAA93D6762

P478

Q42138750-BAC54690-D415-417E-8660-B7445973F1BA

P50

Q42138750-15656C23-B677-44CA-AB4D-CDAD0E1A049D

Q42138750-CD3F8CBA-48A4-466C-A08D-9789C425448F

Q42138750-EBD26A12-FE32-4D3A-843B-D36675FF59AE

P577

Q42138750-30E4073C-46F7-4DC4-9B2D-61F8ACEEBF5F

P5875

Q42138750-FF3AB5C2-0DC8-43A1-B5BB-28DE1485BB54

P698

Q42138750-532B7E8E-8663-49A7-B2AB-7AF7CFA4A076

P921

Q42138750-CF33FC3B-8415-4692-9897-DC91FFAF9D87

P932

Q42138750-ACA6F911-BE04-48A4-A89A-CC63CF38EEAE

P356

P1433

Molecular Therapy

P1476

Loss of miR-29 in myoblasts contributes to dystrophic muscle pathogenesis

@en

P2093

Denis C Guttridge

http://www.w3.org/2001/XMLSchema#string

Huating Wang

http://www.w3.org/2001/XMLSchema#string

Leina Lu

http://www.w3.org/2001/XMLSchema#string

Lijun Wang

http://www.w3.org/2001/XMLSchema#string

Peiyong Jiang

http://www.w3.org/2001/XMLSchema#string

Xiaona Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Utrophin-dystrophin-deficient mice as a model for Duchenne muscular dystrophy

Transcript assembly and quantification by RNA-Seq reveals unannotated transcripts and isoform switching during cell differentiation

Systemic delivery of synthetic microRNA-16 inhibits the growth of metastatic prostate tumors via downregulation of multiple cell-cycle genes

MicroRNA expression in response to murine myocardial infarction: miR-21 regulates fibroblast metalloprotease-2 via phosphatase and tensin homologue

TopHat: discovering splice junctions with RNA-Seq

RNA-seq: an assessment of technical reproducibility and comparison with gene expression arrays

MicroRNA 29c is down-regulated in nasopharyngeal carcinomas, up-regulating mRNAs encoding extracellular matrix proteins.

Human RISC couples microRNA biogenesis and posttranscriptional gene silencing

Development of a lung cancer therapeutic based on the tumor suppressor microRNA-34

MicroRNA-21 contributes to myocardial disease by stimulating MAP kinase signalling in fibroblasts

P304

1222-1233

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/MT.2012.35

http://www.w3.org/2001/XMLSchema#string

P433

6

http://www.w3.org/2001/XMLSchema#string

P478

20

http://www.w3.org/2001/XMLSchema#string

P50

P577

2012-03-20T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

221877659

http://www.w3.org/2001/XMLSchema#string

P698

22434133

http://www.w3.org/2001/XMLSchema#string

P921

P932

3369280

http://www.w3.org/2001/XMLSchema#string