A mouse model of vitiligo with focused epidermal depigmentation requires IFN-γ for autoreactive CD8⁺ T-cell accumulation in the skin. - Wikidata - awgldk - TriplyDB

A mouse model of vitiligo with focused epidermal depigmentation requires IFN-γ for autoreactive CD8⁺ T-cell accumulation in the skin.

A mouse model of vitiligo with focused epidermal depigmentation requires IFN-γ for autoreactive CD8⁺ T-cell accumulation in the skin.

http://www.wikidata.org/entity/Q42145851

about

Bone Marrow T Cells and the Integrated Functions of Recirculating and Tissue-Resident Memory T Cells A central role for inducible heat-shock protein 70 in autoimmune vitiligo Recent advances in understanding vitiligo Immune responses in a mouse model of vitiligo with spontaneous epidermal de- and repigmentation.CD49a Expression Defines Tissue-Resident CD8+ T Cells Poised for Cytotoxic Function in Human Skin.CXCL10 is critical for the progression and maintenance of depigmentation in a mouse model of vitiligo.Adipose-derived stem cells improve efficacy of melanocyte transplantation in animal skin.MHC class II super-enhancer increases surface expression of HLA-DR and HLA-DQ and affects cytokine production in autoimmune vitiligo.Vitiligo road map.Transcriptome analysis reveals markers of aberrantly activated innate immunity in vitiligo lesional and non-lesional skin.Interferon-γ induces senescence in normal human melanocytes.Simvastatin prevents and reverses depigmentation in a mouse model of vitiligo Association of IFN-γ : IL-10 Cytokine Ratio with Nonsegmental Vitiligo Pathogenesis Tumour necrosis factor-α inhibition can stabilize disease in progressive vitiligo.Pediatric Patients with Vitiligo in Eastern China: Abnormalities in 145 Cases Based on Thyroid Function Tests and Immunological Findings Cellular stress and innate inflammation in organ-specific autoimmunity: lessons learned from vitiligo.Divergent effects of T cell costimulation and inflammatory cytokine production on autoimmune peripheral neuropathy provoked by Aire deficiency.Vitiligo blood transcriptomics provides new insights into disease mechanisms and identifies potential novel therapeutic targets.A quantitative increase in regulatory T cells controls development of vitiligo.Innate immune mechanisms in vitiligo: danger from within.Alternative Systemic Treatments for Vitiligo: A Review.Understanding mechanisms of autoimmunity through translational research in vitiligo.Understanding autoimmunity of vitiligo and alopecia areata.Interactome analysis of gene expression profile reveals potential novel key transcriptional regulators of skin pathology in vitiligo.JAK inhibitors in dermatology: The promise of a new drug class.Vitiligo Pathogenesis and Emerging Treatments.Melanocyte-specific CD8+ T cells are associated with epidermal depigmentation in a novel mouse model of vitiligo.Vitiligo: Focus on Clinical Aspects, Immunopathogenesis, and Therapy.T-cell reconstitution after thymus xenotransplantation induces hair depigmentation and loss Isolation of Infiltrating Leukocytes from Mouse Skin Using Enzymatic Digest and Gradient Separation.Increased expression of CXCR3 and its ligands in patients with vitiligo and CXCL10 as a potential clinical marker for vitiligo.The role of the Th1 chemokine CXCL10 in vitiligo.Differential expression of mRNA for T-bet and GATA-3 transcription factors in peripheral blood mononuclear cells of patients with vitiligo.Vitiligo and alopecia areata: apples and oranges?Combination central tolerance and peripheral checkpoint blockade unleashes antimelanoma immunity.IFN-γ inhibits basal and α-MSH-induced melanogenesis.Treatment of vitiligo vulgaris with the combination therapy of topical steroid and vitamin D3 compound.Interfering with the IFN-γ/CXCL10 pathway to develop new targeted treatments for vitiligo.MicroRNA expression profiling identifies potential serum biomarkers for non-segmental vitiligo.Clinical features and histological findings are potential indicators of activity in lesions of common vitiligo.

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A mouse model of vitiligo with focused epidermal depigmentation requires IFN-γ for autoreactive CD8⁺ T-cell accumulation in the skin.

http://www.wikidata.org/entity/Q42145851

description

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Journal of Investigative Dermatology

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E John Wherry

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John E Harris

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Laurence A Turka

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P2860

CD8+ T cell immunity against a tumor/self-antigen is augmented by CD4+ T helper cells and hindered by naturally occurring T regulatory cells.

Mechanisms of spatial and temporal development of autoimmune vitiligo in tyrosinase-specific TCR transgenic mice.

Cytotoxic T cells.

Antigen presentation by the endothelium: a green light for antigen-specific T cell trafficking?

Lymphocytes, neuropeptides, and genes involved in alopecia areata.

Random migration precedes stable target cell interactions of tumor-infiltrating T cells.

Tumor regression and autoimmunity after reversal of a functionally tolerant state of self-reactive CD8+ T cells.

Murine cutaneous mastocytosis and epidermal melanocytosis induced by keratinocyte expression of transgenic stem cell factor.

gp100/pmel 17 is a murine tumor rejection antigen: induction of "self"-reactive, tumoricidal T cells using high-affinity, altered peptide ligand

Interstitial leukocyte migration and immune function.

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10.1038/JID.2011.463

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221798668

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