Distinct gene-expression profiles associated with the susceptibility of pathogen-specific CD4 T cells to HIV-1 infection.
about
New Insights about Treg and Th17 Cells in HIV Infection and Disease ProgressionOropharyngeal Candidiasis in HIV Infection: Analysis of Impaired Mucosal Immune Response to Candida albicans in Mice Expressing the HIV-1 TransgeneImmune restoration after antiretroviral therapy: the pitfalls of hasty or incomplete repairs.Preferential infection of human Ad5-specific CD4 T cells by HIV in Ad5 naturally exposed and recombinant Ad5-HIV vaccinated individualsDefective IL-17- and IL-22-dependent mucosal host response to Candida albicans determines susceptibility to oral candidiasis in mice expressing the HIV-1 transgene.Sequential Dysfunction and Progressive Depletion of Candida albicans-Specific CD4 T Cell Response in HIV-1 Infection.Differences in antigen-specific CD4+ responses to opportunistic infections in HIV infection.Identification of novel HIV-1 dependency factors in primary CCR4(+)CCR6(+)Th17 cells via a genome-wide transcriptional approach.Paediatric HIV infection in the 'omics era: defining transcriptional signatures of viral control and vaccine responses.HIV Skews the Lineage-Defining Transcriptional Profile of Mycobacterium tuberculosis-Specific CD4+ T Cells.Preferential HIV infection of CCR6+ Th17 cells is associated with higher levels of virus receptor expression and lack of CCR5 ligands.T cell susceptibility to HIV influences outcome of opportunistic infections.HIV persists in CCR6+CD4+ T cells from colon and blood during antiretroviral therapy.Transcriptional profiling reveals molecular signatures associated with HIV permissiveness in Th1Th17 cells and identifies peroxisome proliferator-activated receptor gamma as an intrinsic negative regulator of viral replication.A systems framework for vaccine design.Loss of immune control in HIV-infected patients: how does mucosal candidiasis occur?The dual CCR5 and CCR2 inhibitor cenicriviroc does not redistribute HIV into extracellular space: implications for plasma viral load and intracellular DNA decline.HIV Susceptibility of human antigen-specific CD4 T cells in AIDS pathogenesis and vaccine response.HIV-1 Infection Dysregulates Cell Cycle Regulatory Protein p21 in CD4+ T Cells Through miR-20a and miR-106b Regulation.Th17 cells differentiated with mycelial membranes of Candida albicans prevent oral candidiasis.Distinct susceptibility of HIV vaccine vector-induced CD4 T cells to HIV infection.
P2860
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P2860
Distinct gene-expression profiles associated with the susceptibility of pathogen-specific CD4 T cells to HIV-1 infection.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
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2012年论文
@zh-cn
name
Distinct gene-expression profi ...... D4 T cells to HIV-1 infection.
@en
Distinct gene-expression profi ...... D4 T cells to HIV-1 infection.
@nl
type
label
Distinct gene-expression profi ...... D4 T cells to HIV-1 infection.
@en
Distinct gene-expression profi ...... D4 T cells to HIV-1 infection.
@nl
prefLabel
Distinct gene-expression profi ...... D4 T cells to HIV-1 infection.
@en
Distinct gene-expression profi ...... D4 T cells to HIV-1 infection.
@nl
P2093
P2860
P50
P1433
P1476
Distinct gene-expression profi ...... CD4 T cells to HIV-1 infection
@en
P2093
Agnes-Laurence Chenine
Camila Macedo
Martin Nau
Mary Marovich
Maryanne Vahey
Z John Daye
P2860
P304
P356
10.1182/BLOOD-2012-07-446278
P407
P577
2012-12-20T00:00:00Z