Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

about

Ascorbic acid in the prevention and treatment of cancer.Evaluation of the antiaggregant activity of ascorbyl phenolic esters with antioxidant properties.

P2860

Q39031234-18065F8A-7BEA-47FA-9F13-E748D18ED966 Q54265369-8B9944C8-A190-41BE-9726-2C7DFC72B1FC

P2860

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

Item

http://www.wikidata.org/entity/Q42148903

description

2014 nî lūn-bûn

@nan

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

2014年论文

@zh

2014年论文

@zh-cn

name

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@en

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@nl

type

Item

label

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@en

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@nl

prefLabel

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@en

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@nl

P1476

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

@en

P2093

Bruce C Baguley

http://www.w3.org/2001/XMLSchema#string

Emma Richardson

http://www.w3.org/2001/XMLSchema#string

Qi Ding

http://www.w3.org/2001/XMLSchema#string

P2860

Supplemental ascorbate in the supportive treatment of cancer: Prolongation of survival times in terminal human cancer

Supplemental ascorbate in the supportive treatment of cancer: reevaluation of prolongation of survival times in terminal human cancer

Pharmacologic doses of ascorbate act as a prooxidant and decrease growth of aggressive tumor xenografts in mice

Sodium-dependent ascorbic acid transporter family SLC23

Relationship between tumour endothelial cell apoptosis and tumour blood flow shutdown following treatment with the antivascular agent DMXAA in mice.

Mechanisms of tumor vascular shutdown induced by 5,6-dimethylxanthenone-4-acetic acid (DMXAA): Increased tumor vascular permeability.

Vitamin C pharmacokinetics: implications for oral and intravenous use.

A structural approach into human tryptophan hydroxylase and its implications for the regulation of serotonin biosynthesis.

Review of high-dose intravenous vitamin C as an anticancer agent.

Antivascular therapy of cancer: DMXAA.

P304

310

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3389/FONC.2014.00310

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2014-11-05T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

268790549

http://www.w3.org/2001/XMLSchema#string

P698

25414833

http://www.w3.org/2001/XMLSchema#string

P921

vitamin C

P932

4220656

http://www.w3.org/2001/XMLSchema#string

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

about

P2860

P2860

Preliminary Evidence That High-Dose Vitamin C has a Vascular Disrupting Action in Mice.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P478

P577

P5875

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P478

P577

P5875

P698

P921

P932