Complement receptor Mac-1 is an adaptor for NB1 (CD177)-mediated PR3-ANCA neutrophil activation.
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The human neutrophil subsets defined by the presence or absence of OLFM4 both transmigrate into tissue in vivo and give rise to distinct NETs in vitroLy6 family proteins in neutrophil biologyThe neutrophil in antineutrophil cytoplasmic autoantibody-associated vasculitisGenetic mechanism of human neutrophil antigen 2 deficiency and expression variationsExtracellular protein disulfide isomerase regulates ligand-binding activity of αMβ2 integrin and neutrophil recruitment during vascular inflammationImmunomodulation with eicosapentaenoic acid supports the treatment of autoimmune small-vessel vasculitis.Characterization of a novel mouse model with genetic deletion of CD177Intermediate monocytes in ANCA vasculitis: increased surface expression of ANCA autoantigens and IL-1β secretion in response to anti-MPO antibodiesPhenotypic and functional plasticity of cells of innate immunity: macrophages, mast cells and neutrophils.Ly6G ligation blocks recruitment of neutrophils via a β2-integrin-dependent mechanismEndothelium-neutrophil interactions in ANCA-associated diseasesATG4B/autophagin-1 regulates intestinal homeostasis and protects mice from experimental colitis.Anti-myeloperoxidase antibodies attenuate the monocyte response to LPS and shape macrophage development.Pathogenesis of antineutrophil cytoplasmic autoantibody vasculitis.How anti-neutrophil cytoplasmic autoantibodies activate neutrophils.Neutrophils come of age in chronic inflammation.Neutrophil proteinase 3 and dipeptidyl peptidase I (cathepsin C) as pharmacological targets in granulomatosis with polyangiitis (Wegener granulomatosis).GSL-enriched membrane microdomains in innate immune responses.Folate Receptor β Regulates Integrin CD11b/CD18 Adhesion of a Macrophage Subset to Collagen.Neutral serine proteases of neutrophils.Characterization of the CD177 interaction with the ANCA antigen proteinase 3.CD177 modulates human neutrophil migration through activation-mediated integrin and chemoreceptor regulation.Gene silencing and a novel monoallelic expression pattern in distinct CD177 neutrophil subsets.The neutrophil: one cell on many missions or many cells with different agendas?The Road Traveled: Genomics and Biomarkers in Antineutrophil Cytoplasmic Antibody-Associated Vasculitis.Interaction between CD177 and platelet endothelial cell adhesion molecule-1 downregulates membrane-bound proteinase-3 (PR3) expression on neutrophils and attenuates neutrophil activation induced by PR3-ANCA
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Complement receptor Mac-1 is an adaptor for NB1 (CD177)-mediated PR3-ANCA neutrophil activation.
description
2010 nî lūn-bûn
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2010年の論文
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2010年学术文章
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2010年学术文章
@zh-my
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@zh-sg
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name
Complement receptor Mac-1 is a ...... R3-ANCA neutrophil activation.
@en
Complement receptor Mac-1 is an adaptor for NB1
@nl
type
label
Complement receptor Mac-1 is a ...... R3-ANCA neutrophil activation.
@en
Complement receptor Mac-1 is an adaptor for NB1
@nl
prefLabel
Complement receptor Mac-1 is a ...... R3-ANCA neutrophil activation.
@en
Complement receptor Mac-1 is an adaptor for NB1
@nl
P2093
P2860
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Complement receptor Mac-1 is a ...... R3-ANCA neutrophil activation.
@en
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Anje Sporbert
Behnaz Bayat
Susanne Rolle
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P304
P356
10.1074/JBC.M110.171256
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P577
2010-12-30T00:00:00Z