Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria.

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Opsonization of malaria-infected erythrocytes activates the inflammasome and enhances inflammatory cytokine secretion by human macrophages.IRAK Inhibitor Protects the Intestinal Tract of Necrotizing Enterocolitis by Inhibiting the Toll-Like Receptor (TLR) Inflammatory Signaling Pathway in Rats.

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Q36483344-B006D541-2FFC-40A4-8FD1-A1955C064750 Q54108257-63709AB7-0A64-404C-A3B1-4A282F4BF3EB

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Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria.

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2010 nî lūn-bûn

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2010年の論文

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2010年学术文章

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2010年学术文章

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2010年学术文章

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2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

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2010年學術文章

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2010年學術文章

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Divergent roles of IRAK4-media ...... ntal models of severe malaria.

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Divergent roles of IRAK4-media ...... ntal models of severe malaria.

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type

Item

label

Divergent roles of IRAK4-media ...... ntal models of severe malaria.

@en

Divergent roles of IRAK4-media ...... ntal models of severe malaria.

@nl

prefLabel

Divergent roles of IRAK4-media ...... ntal models of severe malaria.

@en

Divergent roles of IRAK4-media ...... ntal models of severe malaria.

@nl

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Divergent roles of IRAK4-media ...... ntal models of severe malaria.

@en

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Constance A M Finney

http://www.w3.org/2001/XMLSchema#string

Michael Hawkes

http://www.w3.org/2001/XMLSchema#string

W Conrad Liles

http://www.w3.org/2001/XMLSchema#string

Wen-Chen Yeh

http://www.w3.org/2001/XMLSchema#string

Ziyue Lu

http://www.w3.org/2001/XMLSchema#string

P2860

Pyogenic bacterial infections in humans with IRAK-4 deficiency

Toll-like receptor (TLR) polymorphisms in African children: Common TLR-4 variants predispose to severe malaria.

Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity

A Mal functional variant is associated with protection against invasive pneumococcal disease, bacteremia, malaria and tuberculosis

Severe falciparum malaria. World Health Organization, Communicable Diseases Cluster

Toll-like receptors and innate immunity

Tumor-necrosis factor and other cytokines in cerebral malaria: experimental and clinical data.

Artesunate versus quinine for treatment of severe falciparum malaria: a randomised trial.

Murine cerebral malaria development is independent of toll-like receptor signaling

CD36 and TLR interactions in inflammation and phagocytosis: implications for malaria.

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69-74

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scholarly article

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10.4269/AJTMH.2010.09-0753

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English

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http://www.w3.org/2001/XMLSchema#string

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Kevin C Kain

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2010-07-01T00:00:00Z

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44901975

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20595480

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malaria

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2912578

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Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria.

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P2860

P2860

Divergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria.

description

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type

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P1433

P1476

P2093

P2860

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P356

P407

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P5875

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P1476

P2093

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P356

P407

P433

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P932