Cerebral endothelial activation and signal transduction mechanisms during inflammation and infectious disease.

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Cerebral endothelial activation and signal transduction mechanisms during inflammation and infectious disease.

Item

http://www.wikidata.org/entity/Q42157874

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1999 nî lūn-bûn

@nan

1999年の論文

@ja

1999年学术文章

@wuu

1999年学术文章

@zh-cn

1999年学术文章

@zh-hans

1999年学术文章

@zh-my

1999年学术文章

@zh-sg

1999年學術文章

@yue

1999年學術文章

@zh

1999年學術文章

@zh-hant

name

Cerebral endothelial activatio ...... mation and infectious disease.

@en

Cerebral endothelial activatio ...... mation and infectious disease.

@nl

type

Item

label

Cerebral endothelial activatio ...... mation and infectious disease.

@en

Cerebral endothelial activatio ...... mation and infectious disease.

@nl

prefLabel

Cerebral endothelial activatio ...... mation and infectious disease.

@en

Cerebral endothelial activatio ...... mation and infectious disease.

@nl

P1476

Cerebral endothelial activatio ...... mation and infectious disease.

@en

P2093

Kalaria RN

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P2860

Challenging cytokine redundancy: inflammatory cell movement and clinical course of experimental autoimmune encephalomyelitis are normal in lymphotoxin-deficient, but not tumor necrosis factor-deficient, mice

The Jak-STAT pathway: cytokine signalling from the receptor to the nucleus.

Interferon-gamma receptor-mediated but not tumor necrosis factor receptor type 1- or type 2-mediated signaling is crucial for the activation of cerebral blood vessel endothelial cells and microglia in murine Toxoplasma encephalitis.

Effects of interferon-gamma on primary cultures of human brain microvessel endothelial cells

Effects of protein tyrosine kinase inhibitors on cytokine-induced adhesion molecule expression by human umbilical vein endothelial cells

Microglia stimulate naive T-cell differentiation without stimulating T-cell proliferation.

Protein kinase C activation is involved in ultraviolet B irradiation-induced endothelial cell ICAM-1 up-regulation and lymphocyte-endothelium interaction in vitro.

Detailed in vivo analysis of interferon-gamma induced major histocompatibility complex expression in the the central nervous system: astrocytes fail to express major histocompatibility complex class I and II molecules.

Differential reactivity of brain microvascular endothelial cells to TNF reflects the genetic susceptibility to cerebral malaria.

Expression of major histocompatibility complex antigens on mouse brain microvascular endothelial cells in relation to susceptibility to cerebral malaria.

P304

1311-1314

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scholarly article

editorial

P356

10.1016/S0002-9440(10)65383-7

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P407

English

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P478

154

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P577

1999-05-01T00:00:00Z

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P698

10329582

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P921

endothelium

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1866610

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