Transcription factor Nrf2 is protective during ischemic and nephrotoxic acute kidney injury in mice.
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Effect of redox modulating NRF2 activators on chronic kidney diseaseNRF2 Is a Key Target for Prevention of Noise-Induced Hearing Loss by Reducing Oxidative Damage of Cochlea.Endogenous hydrogen sulfide production is essential for dietary restriction benefitsmiR-125b transcriptionally increased by Nrf2 inhibits AhR repressor, which protects kidney from cisplatin-induced injuryMeta-analysis of molecular response of kidney to ischemia reperfusion injury for the identification of new candidate genesThe protective role of Nrf2 in streptozotocin-induced diabetic nephropathy.Suppressed autophagic response underlies augmentation of renal ischemia/reperfusion injury by type 2 diabetes.Baicalin ameliorates H2O2 induced cytotoxicity in HK-2 cells through the inhibition of ER stress and the activation of Nrf2 signalingMicroRNA-34a is induced via p53 during cisplatin nephrotoxicity and contributes to cell survival.Transcriptional regulation of renal cytoprotective genes by Nrf2 and its potential use as a therapeutic target to mitigate cisplatin-induced nephrotoxicity.Bardoxolone methyl (CDDO-Me) as a therapeutic agent: an update on its pharmacokinetic and pharmacodynamic properties.Activating Nrf-2 signaling depresses unilateral ureteral obstruction-evoked mitochondrial stress-related autophagy, apoptosis and pyroptosis in kidney.Bardoxolone methyl (BARD) ameliorates ischemic AKI and increases expression of protective genes Nrf2, PPARĪ³, and HO-1.N-acetylcysteine is effective for prevention but not for treatment of folic acid-induced acute kidney injury in micePoly(ADP-ribose) polymerase-1 is a key mediator of cisplatin-induced kidney inflammation and injury.Proteomic analysis of 3-MCPD and 3-MCPD dipalmitate-induced toxicity in rat kidney.Reactive Oxygen Species and Nuclear Factor Erythroid 2-Related Factor 2 Activation in Diabetic Nephropathy: A Hidden TargetActivation of the Nrf2 Cell Defense Pathway by Ancient Foods: Disease Prevention by Important Molecules and Microbes Lost from the Modern Western Diet."Curcumin, the King of Spices": Epigenetic Regulatory Mechanisms in the Prevention of Cancer, Neurological, and Inflammatory Diseases.Anti-inflammatory activity of Barleria lupulina: Identification of active compounds that activate the Nrf2 cell defense pathway, organize cortical actin, reduce stress fibers, and improve cell junctions in microvascular endothelial cells.Diabetic Wound Healing and Activation of Nrf2 by Herbal MedicineSynthetic oleanane triterpenoids: multifunctional drugs with a broad range of applications for prevention and treatment of chronic disease.T Lymphocyte-Specific Activation of Nrf2 Protects from AKI.Integrated transcriptomic and proteomic analyses uncover regulatory roles of Nrf2 in the kidney.Overactivation of the nuclear factor (erythroid-derived 2)-like 2-antioxidant response element pathway in hepatocytes decreases hepatic ischemia/reperfusion injury in mice.Mouse model of ischemic acute kidney injury: technical notes and tricksDisruption of Type-I IFN pathway ameliorates preservation damage in mouse orthotopic liver transplantation via HO-1 dependent mechanism.Targeting the transcription factor Nrf2 to ameliorate oxidative stress and inflammation in chronic kidney diseaseNrf2: a potential target for new therapeutics in liver disease.Kidney epithelium specific deletion of kelch-like ECH-associated protein 1 (Keap1) causes hydronephrosis in mice.Role of impaired Nrf2 activation in the pathogenesis of oxidative stress and inflammation in chronic tubulo-interstitial nephropathy.Protective Effects of Berberine on Renal Injury in Streptozotocin (STZ)-Induced Diabetic Mice.Pathophysiology of acute kidney injuryThe Nrf2 triterpenoid activator, CDDO-imidazolide, protects kidneys from ischemia-reperfusion injury in mice.Keap1 inhibition attenuates glomerulosclerosis.Tubular Peroxiredoxin 3 as a Predictor of Renal Recovery from Acute Tubular Necrosis in Patients with Chronic Kidney Disease.Nrf2-ARE stress response mechanism: a control point in oxidative stress-mediated dysfunctions and chronic inflammatory diseases.Hypoxia. 1. Intracellular sensors for oxygen and oxidative stress: novel therapeutic targets.New era for drug discovery and development in renal disease.The Nrf2 pathway in the progression of renal disease.
P2860
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P2860
Transcription factor Nrf2 is protective during ischemic and nephrotoxic acute kidney injury in mice.
description
2009 nĆ® lÅ«n-bĆ»n
@nan
2009幓ć®č«ę
@ja
2009幓č«ę
@yue
2009幓č«ę
@zh-hant
2009幓č«ę
@zh-hk
2009幓č«ę
@zh-mo
2009幓č«ę
@zh-tw
2009幓č®ŗę
@wuu
2009幓č®ŗę
@zh
2009幓č®ŗę
@zh-cn
name
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@en
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@nl
type
label
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@en
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@nl
prefLabel
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@en
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@nl
P2093
P356
P1433
P1476
Transcription factor Nrf2 is p ...... c acute kidney injury in mice.
@en
P2093
Dmitry N Grigoryev
Hamid Rabb
Manchang Liu
Masayuki Yamamoto
Michael T Crow
Sekhar P Reddy
P2888
P304
P356
10.1038/KI.2009.157
P407
P577
2009-05-13T00:00:00Z