Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLD.
about
Role of inflammatory response in liver diseases: Therapeutic strategies.Role of Oxidative Stress in Pathophysiology of Nonalcoholic Fatty Liver Disease.Interaction of volatile organic compounds and underlying liver disease: a new paradigm for riskAdapted Immune Responses of Myeloid-Derived Cells in Fatty Liver Disease
P2860
Lack of NLRP3-inflammasome leads to gut-liver axis derangement, gut dysbiosis and a worsened phenotype in a mouse model of NAFLD.
description
2017 nî lūn-bûn
@nan
2017年の論文
@ja
2017年論文
@yue
2017年論文
@zh-hant
2017年論文
@zh-hk
2017年論文
@zh-mo
2017年論文
@zh-tw
2017年论文
@wuu
2017年论文
@zh
2017年论文
@zh-cn
name
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@en
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@nl
type
label
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@en
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@nl
prefLabel
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@en
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@nl
P2093
P2860
P50
P1433
P1476
Lack of NLRP3-inflammasome lea ...... ype in a mouse model of NAFLD.
@en
P2093
Antonio Giordano
Chiara Rychlicki
Claudio Pinto
Cristina Fraumene
Debora Maria Giordano
Eleonora Mingarelli
Emma Buzzigoli
Irene Pierantonelli
Laura Agostinelli
Loris Sartini
P2860
P2888
P356
10.1038/S41598-017-11744-6
P407
P577
2017-09-22T00:00:00Z
P6179
1091849468