Intrinsic, Pro-Apoptotic Effects of IGFBP-3 on Breast Cancer Cells are Reversible: Involvement of PKA, Rho, and Ceramide.

about

IGFBP-3 inhibits TNF-α production and TNFR-2 signaling to protect against retinal endothelial cell apoptosis.Insulin-like growth factor binding protein-3 (IGFBP-3): Novel ligands mediate unexpected functions.New insights on the Intrinsic, Pro-Apoptotic Effect of IGFB3 in Breast Cancer.

P2860

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P2860

Intrinsic, Pro-Apoptotic Effects of IGFBP-3 on Breast Cancer Cells are Reversible: Involvement of PKA, Rho, and Ceramide.

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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type

Item

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Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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P1476

Intrinsic, Pro-Apoptotic Effec ...... ent of PKA, Rho, and Ceramide.

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P2093

Jeff M P Holly

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P2860

Insulin-like growth factor-independent effects mediated by a C-terminal metal-binding domain of insulin-like growth factor binding protein-3

Insulin-like growth factor binding protein 1 stimulates cell migration and binds to the alpha 5 beta 1 integrin by means of its Arg-Gly-Asp sequence

Identification of a novel cell death receptor mediating IGFBP-3-induced anti-tumor effects in breast and prostate cancer

The small GTP-binding protein rho regulates the assembly of focal adhesions and actin stress fibers in response to growth factors

Direct functional interactions between insulin-like growth factor-binding protein-3 and retinoid X receptor-alpha regulate transcriptional signaling and apoptosis

Cellular internalization of insulin-like growth factor binding protein-3: distinct endocytic pathways facilitate re-uptake and nuclear localization

Insulin-like growth factor-binding protein (IGFBP-3) predisposes breast cancer cells to programmed cell death in a non-IGF-dependent manner

Potentiation of growth factor signaling by insulin-like growth factor-binding protein-3 in breast epithelial cells requires sphingosine kinase activity

Dual effects of IGFBP-3 on endothelial cell apoptosis and survival: involvement of the sphingolipid signaling pathways

Integrins: emerging paradigms of signal transduction

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scholarly article

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10.3389/FENDO.2011.00013

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Claire M Perks

Carla Burrows

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2011-05-16T00:00:00Z

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apoptotic process

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Intrinsic, Pro-Apoptotic Effects of IGFBP-3 on Breast Cancer Cells are Reversible: Involvement of PKA, Rho, and Ceramide.

about

P2860

P2860

Intrinsic, Pro-Apoptotic Effects of IGFBP-3 on Breast Cancer Cells are Reversible: Involvement of PKA, Rho, and Ceramide.

description

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