Selection of autophagy or apoptosis in cells exposed to ER-stress depends on ATF4 expression pattern with or without CHOP expression. - Wikidata - awgldk - TriplyDB

Selection of autophagy or apoptosis in cells exposed to ER-stress depends on ATF4 expression pattern with or without CHOP expression.

Selection of autophagy or apoptosis in cells exposed to ER-stress depends on ATF4 expression pattern with or without CHOP expression.

http://www.wikidata.org/entity/Q42175772

about

Conserved and plant-unique strategies for overcoming endoplasmic reticulum stress Shiga toxins induce autophagic cell death in intestinal epithelial cells via the endoplasmic reticulum stress pathway.Autophagy is involved in ethanol-induced cardia bifida during chick cardiogenesis.The endoplasmic reticulum, not the pH gradient, drives calcium refilling of lysosomes.Lithocholic acid induces endoplasmic reticulum stress, autophagy and mitochondrial dysfunction in human prostate cancer cells.Sustained HSP25 Expression Induces Clasmatodendrosis via ER Stress in the Rat Hippocampus.Bisdemethoxycurcumin exerts pro-apoptotic effects in human pancreatic adenocarcinoma cells through mitochondrial dysfunction and a GRP78-dependent pathway.Impaired autophagic flux is associated with increased endoplasmic reticulum stress during the development of NAFLD.The Cytotoxicity of the Ajoene Analogue BisPMB in WHCO1 Oesophageal Cancer Cells Is Mediated by CHOP/GADD153.Bufalin induces the interplay between apoptosis and autophagy in glioma cells through endoplasmic reticulum stress.Endoplasmic Reticulum (ER) Stress and Endocrine Disorders.Subtilase cytotoxin produced by locus of enterocyte effacement-negative Shiga-toxigenic Escherichia coli induces stress granule formation.Zbtb38 is a novel target for spinal cord injury.Glycolaldehyde induces endoplasmic reticulum stress and apoptosis in Schwann cells EIF2S3 Mutations Associated with Severe X-Linked Intellectual Disability Syndrome MEHMO.An ATF4-Signal-Modulating Machine Other Than GADD34 Acts in ATF4-to-CHOP Signaling to Block CHOP Expression in ER-Stress-Related Autophagy.Surviving Stress: Modulation of ATF4-Mediated Stress Responses in Normal and Malignant Cells.Early Neurodegeneration in the Brain of a Child Without Functional PKR-like Endoplasmic Reticulum Kinase.P2RX7-MAPK1/2-SP1 axis inhibits MTOR independent HSPB1-mediated astroglial autophagy.7-Acetylsinumaximol B Induces Apoptosis and Autophagy in Human Gastric Carcinoma Cells through Mitochondria Dysfunction and Activation of the PERK/eIF2α/ATF4/CHOP Signaling Pathway.

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P2860

Selection of autophagy or apoptosis in cells exposed to ER-stress depends on ATF4 expression pattern with or without CHOP expression.

http://www.wikidata.org/entity/Q42175772

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Selection of autophagy or apop ...... th or without CHOP expression.

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Selection of autophagy or apop ...... th or without CHOP expression.

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type

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Selection of autophagy or apop ...... th or without CHOP expression.

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Selection of autophagy or apop ...... th or without CHOP expression.

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prefLabel

Selection of autophagy or apop ...... th or without CHOP expression.

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Selection of autophagy or apop ...... th or without CHOP expression.

@nl

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Selection of autophagy or apop ...... th or without CHOP expression.

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P2093

Hiroki Matsumoto

http://www.w3.org/2001/XMLSchema#string

Hiroshi Nakagawa

http://www.w3.org/2001/XMLSchema#string

Kazuhiko Nishimura

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Makoto Kawano

http://www.w3.org/2001/XMLSchema#string

Masayuki Takeda

http://www.w3.org/2001/XMLSchema#string

Saburo Matsuo

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Satoshi Matsuyama

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Shuichi Miyazaki

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P2860

Regulation of protein synthesis by hypoxia via activation of the endoplasmic reticulum kinase PERK and phosphorylation of the translation initiation factor eIF2alpha.

Autophagy: molecular machinery for self-eating

Signal integration in the endoplasmic reticulum unfolded protein response

Protein translation and folding are coupled by an endoplasmic-reticulum-resident kinase

CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum

An integrated stress response regulates amino acid metabolism and resistance to oxidative stress

The role of autophagy during the early neonatal starvation period

Autophagy is activated for cell survival after endoplasmic reticulum stress

Role of Bcl-2 family proteins in a non-apoptotic programmed cell death dependent on autophagy genes

The unfolded protein response protects human tumor cells during hypoxia through regulation of the autophagy genes MAP1LC3B and ATG5.

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1084-1090

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10.1242/BIO.20135033

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10

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2

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24167719

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3798192

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