Deficiency of ATP-binding cassette transporters A1 and G1 in macrophages increases inflammation and accelerates atherosclerosis in mice. - Wikidata - awgldk - TriplyDB

Deficiency of ATP-binding cassette transporters A1 and G1 in macrophages increases inflammation and accelerates atherosclerosis in mice.

Deficiency of ATP-binding cassette transporters A1 and G1 in macrophages increases inflammation and accelerates atherosclerosis in mice.

http://www.wikidata.org/entity/Q42186976

about

Advances in the Study of the Antiatherogenic Function and Novel Therapies for HDL Cholesterol efflux pathways regulate myelopoiesis: a potential link to altered macrophage function in atherosclerosis Macrophage functions in atherosclerosis Cholesterol, inflammation and innate immunity Reactive Oxygen Species (ROS) Mediate p300-dependent STAT1 Protein Interaction with Peroxisome Proliferator-activated Receptor (PPAR)-γ in CD36 Protein Expression and Foam Cell Formation Liver X Receptors Link Lipid Metabolism and Inflammation.LDL cholesterol modulates human CD34+ HSPCs through effects on proliferation and the IL-17 G-CSF axis Allicin induces the upregulation of ABCA1 expression via PPARγ/LXRα signaling in THP-1 macrophage-derived foam cells Disruption of mammalian target of rapamycin complex 1 in macrophages decreases chemokine gene expression and atherosclerosis.Mechanisms that regulate macrophage burden in atherosclerosis.Placental ABCA1 Expression Is Increased in Spontaneous Preterm Deliveries Compared with Iatrogenic Preterm Deliveries and Term Deliveries.Myeloid cell-specific ATP-binding cassette transporter A1 deletion has minimal impact on atherogenesis in atherogenic diet-fed low-density lipoprotein receptor knockout mice.IL-24 gene polymorphisms are associated with cardiometabolic parameters and cardiovascular risk factors but not with premature coronary artery disease: the genetics of atherosclerotic disease Mexican study.microRNAs and HDL life cycle.Regulation and consequences of monocytosis ATP-binding cassette transporter 1 attenuates ovalbumin-induced neutrophilic airway inflammation.Recent developments in vascular biology High-density lipoprotein: a novel target for antirestenosis therapy ABCA1 and nascent HDL biogenesis The mutual interplay of lipid metabolism and the cells of the immune system in relation to atherosclerosis Foam cells and the pathogenesis of kidney disease.LXRs link metabolism to inflammation through Abca1-dependent regulation of membrane composition and TLR signaling.MicroRNA-27 Prevents Atherosclerosis by Suppressing Lipoprotein Lipase-Induced Lipid Accumulation and Inflammatory Response in Apolipoprotein E Knockout Mice Functional analysis and transcriptomic profiling of iPSC-derived macrophages and their application in modeling Mendelian disease.Vitamin D Protects Against Atherosclerosis via Regulation of Cholesterol Efflux and Macrophage Polarization in Hypercholesterolemic Swine.The rs7044343 Polymorphism of the Interleukin 33 Gene Is Associated with Decreased Risk of Developing Premature Coronary Artery Disease and Central Obesity, and Could Be Involved in Regulating the Production of IL-33.ABCA1 agonist peptides for the treatment of disease.Macrophage Phenotype and Function in Different Stages of Atherosclerosis.Association between Polymorphisms and Haplotype in the ABCA1 Gene and Overweight/Obesity Patients in the Uyghur Population of China.Disordered haematopoiesis and athero-thrombosis.Leukocyte Calpain Deficiency Reduces Angiotensin II-Induced Inflammation and Atherosclerosis But Not Abdominal Aortic Aneurysms in Mice.CLI-095 decreases atherosclerosis by modulating foam cell formation in apolipoprotein E-deficient mice.Deficiency of ATP-Binding Cassette Transporters A1 and G1 in Endothelial Cells Accelerates Atherosclerosis in Mice.Subverting sterols: rerouting an oxysterol-signaling pathway to promote tumor growth Systemic and renal lipids in kidney disease development and progression.Clinically used selective estrogen receptor modulators affect different steps of macrophage-specific reverse cholesterol transport.Subjects with low plasma HDL cholesterol levels are characterized by an inflammatory and oxidative phenotype.Increased atherosclerotic lesion formation and vascular leukocyte accumulation in renal impairment are mediated by interleukin-17A.Sphingomyelin depletion impairs anionic phospholipid inward translocation and induces cholesterol efflux.Changes in serum inflammatory markers are associated with changes in apolipoprotein A1 but not B after the initiation of dialysis

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P2860

Deficiency of ATP-binding cassette transporters A1 and G1 in macrophages increases inflammation and accelerates atherosclerosis in mice.

http://www.wikidata.org/entity/Q42186976

description

2013 nî lūn-bûn

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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2013年论文

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2013年论文

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name

Deficiency of ATP-binding cass ...... rates atherosclerosis in mice.

@en

Deficiency of ATP-binding cass ...... rates atherosclerosis in mice.

@nl

type

label

Deficiency of ATP-binding cass ...... rates atherosclerosis in mice.

@en

Deficiency of ATP-binding cass ...... rates atherosclerosis in mice.

@nl

prefLabel

Deficiency of ATP-binding cass ...... rates atherosclerosis in mice.

@en

Deficiency of ATP-binding cass ...... rates atherosclerosis in mice.

@nl

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CIRCRESAHA.113.301086

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Circulation Research

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Deficiency of ATP-binding cass ...... erates atherosclerosis in mice

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Alan R Tall

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Carrie Welch

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Darren J Gorman

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John S Parks

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Marit Westerterp

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Mi Wang

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Mojdeh S Kappus

http://www.w3.org/2001/XMLSchema#string

Nan Wang

http://www.w3.org/2001/XMLSchema#string

Sandra Abramowicz

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Tamara A Pagler

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P2860

Mutations in ABC1 in Tangier disease and familial high-density lipoprotein deficiency

The gene encoding ATP-binding cassette transporter 1 is mutated in Tangier disease

Tangier disease is caused by mutations in the gene encoding ATP-binding cassette transporter 1

ATP-binding cassette transporters G1 and G4 mediate cellular cholesterol efflux to high-density lipoproteins

Niacin in patients with low HDL cholesterol levels receiving intensive statin therapy

Effects of torcetrapib in patients at high risk for coronary events

Targeted inactivation of hepatic Abca1 causes profound hypoalphalipoproteinemia and kidney hypercatabolism of apoA-I

Apo A-I inhibits foam cell formation in Apo E-deficient mice after monocyte adherence to endothelium

Leukocyte ABCA1 controls susceptibility to atherosclerosis and macrophage recruitment into tissues.

Impact of macrophage inflammatory protein-1α deficiency on atherosclerotic lesion formation, hepatic steatosis, and adipose tissue expansion

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10.1161/CIRCRESAHA.113.301086

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11

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112

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Edward A Fisher

Laurent Yvan-Charvet

Prabhakara R Nagareddy

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2013-04-09T00:00:00Z

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23572498

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atherosclerosis

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3839866

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