Rapid surface accumulation of NMDA receptors increases glutamatergic excitation during status epilepticus.
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Plasticity of Hippocampal Excitatory-Inhibitory Balance: Missing the Synaptic Control in the Epileptic BrainCommunity structure analysis of transcriptional networks reveals distinct molecular pathways for early- and late-onset temporal lobe epilepsy with childhood febrile seizuresMolecular mechanisms of epilepsyNeurosteroid-sensitive δ-GABAA receptors: A role in epileptogenesis?Enhanced AMPA receptor-mediated neurotransmission on CA1 pyramidal neurons during status epilepticus.Status Epilepticus.The pervasive reduction of GABA-mediated synaptic inhibition of principal neurons in the hippocampus during status epilepticus.Phosphatase inhibition prevents the activity-dependent trafficking of GABAA receptors during status epilepticus in the young animal.Refractory and super-refractory status epilepticus--an update.NR4A1 Knockdown Suppresses Seizure Activity by Regulating Surface Expression of NR2BSubunit composition of neurotransmitter receptors in the immature and in the epileptic brain.Neuroprotective effects of deep hypothermia in refractory status epilepticusEvidence for TNFα action on excitatory and inhibitory neurotransmission in the central amygdala: a brain site influenced by stressPilocarpine-Induced Status Epilepticus Is Associated with Changes in the Actin-Modulating Protein Synaptopodin and Alterations in Long-Term Potentiation in the Mouse Hippocampus.Regulation of GABAARs by phosphorylationTherapeutic choices in convulsive status epilepticus.Glutamatergic Mechanisms Associated with Seizures and Epilepsy.Pharmacologic treatment of status epilepticus.The epileptic amygdala: Toward the development of a neural prosthesis by temporally coded electrical stimulation.Benzodiazepine-refractory status epilepticus: pathophysiology and principles of treatment.Recent advances in epilepsy.Midazolam-ketamine dual therapy stops cholinergic status epilepticus and reduces Morris water maze deficits.Trafficking of NMDA receptors during status epilepticus: therapeutic implications.Neonatal seizures and disruption to neurotransmitter systems.Intranasal Therapy to Stop Status Epilepticus in Prehospital Settings.Treatment of experimental status epilepticus with synergistic drug combinations.Simultaneous triple therapy for the treatment of status epilepticus.Electroencephalographic Patterns in Neurocritical Care: Pathologic Contributors or Epiphenomena?Down-regulation of Pin1 in Temporal Lobe Epilepsy Patients and Mouse Model.Hippocampal distribution of IL-1β and IL-1RI following lithium-pilocarpine-induced status epilepticus in the developing rat.Role of the Fyn-PKCδ signaling in SE-induced neuroinflammation and epileptogenesis in experimental models of temporal lobe epilepsy.Comparing the Antiseizure and Neuroprotective Efficacy of LY293558, Diazepam, Caramiphen, and LY293558-Caramiphen Combination against Soman in a Rat Model Relevant to the Pediatric Population.Mechanisms of status epilepticus: α-Amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptor hypothesis
P2860
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P2860
Rapid surface accumulation of NMDA receptors increases glutamatergic excitation during status epilepticus.
description
2013 nî lūn-bûn
@nan
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
2013年论文
@zh
2013年论文
@zh-cn
name
Rapid surface accumulation of ...... ion during status epilepticus.
@en
Rapid surface accumulation of ...... ion during status epilepticus.
@nl
type
label
Rapid surface accumulation of ...... ion during status epilepticus.
@en
Rapid surface accumulation of ...... ion during status epilepticus.
@nl
prefLabel
Rapid surface accumulation of ...... ion during status epilepticus.
@en
Rapid surface accumulation of ...... ion during status epilepticus.
@nl
P2093
P2860
P1476
Rapid surface accumulation of ...... ion during status epilepticus.
@en
P2093
Claude G Wasterlain
David E Naylor
Hantao Liu
Jerome Niquet
P2860
P304
P356
10.1016/J.NBD.2012.12.015
P577
2013-01-08T00:00:00Z