C5a/C5aR pathway is essential for the pathogenesis of murine viral fulminant hepatitis by way of potentiating Fgl2/fibroleukin expression. - Wikidata - awgldk - TriplyDB

C5a/C5aR pathway is essential for the pathogenesis of murine viral fulminant hepatitis by way of potentiating Fgl2/fibroleukin expression.

C5a/C5aR pathway is essential for the pathogenesis of murine viral fulminant hepatitis by way of potentiating Fgl2/fibroleukin expression.

http://www.wikidata.org/entity/Q42233208

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Regulation of Stress Responses and Translational Control by Coronavirus A Proof of Concept, Phase II Randomized European Trial, on the Efficacy of ALF-5755, a Novel Extracellular Matrix-Targeted Antioxidant in Patients with Acute Liver Diseases The NLRP3 Inflammasome and IL-1β Accelerate Immunologically Mediated Pathology in Experimental Viral Fulminant Hepatitis.C5b-9 Staining Correlates With Clinical and Tumor Stage in Gastric Adenocarcinoma.C5a/C5aR pathway is essential for up-regulating SphK1 expression through p38-MAPK activation in acute liver failure.The inflammatory response triggered by Influenza virus: a two edged sword.A disparate subset of double-negative T cells contributes to the outcome of murine fulminant viral hepatitis via effector molecule fibrinogen-like protein 2.Macrophage scavenger receptor 1 contributes to pathogenesis of fulminant hepatitis via neutrophil mediated complement activation.Von Willebrand factor protects against acute CCl4-induced hepatotoxicity through phospho-p38 MAPK signaling pathway inhibition.Complement System as a Target for Therapies to Control Liver Regeneration/Damage in Acute Liver Failure Induced by Viral Hepatitis

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P2860

C5a/C5aR pathway is essential for the pathogenesis of murine viral fulminant hepatitis by way of potentiating Fgl2/fibroleukin expression.

http://www.wikidata.org/entity/Q42233208

description

2014 nî lūn-bûn

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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2014年论文

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2014年论文

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name

C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

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C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

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type

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C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

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C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

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C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

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C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

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C5a/C5aR pathway is essential ...... g Fgl2/fibroleukin expression.

@en

P2093

Fei Yang

http://www.w3.org/2001/XMLSchema#string

Gui-lian Xu

http://www.w3.org/2001/XMLSchema#string

Gui-qing Li

http://www.w3.org/2001/XMLSchema#string

Jian Chen

http://www.w3.org/2001/XMLSchema#string

Li-xin Zheng

http://www.w3.org/2001/XMLSchema#string

Yu-zhang Wu

http://www.w3.org/2001/XMLSchema#string

P2860

The orphan receptor C5L2 has high affinity binding sites for complement fragments C5a and C5a des-Arg(74)

Novel homologues of CSBP/p38 MAP kinase: activation, substrate specificity and sensitivity to inhibition by pyridinyl imidazoles

A synthetic inhibitor of the mitogen-activated protein kinase cascade

SB 203580 is a specific inhibitor of a MAP kinase homologue which is stimulated by cellular stresses and interleukin-1

Cloning and functional expression of the canine anaphylatoxin C5a receptor. Evidence for high interspecies variability.

The complement inhibitor eculizumab in paroxysmal nocturnal hemoglobinuria.

The Fgl2/fibroleukin prothrombinase contributes to immunologically mediated thrombosis in experimental and human viral hepatitis

Role of C5a in inflammatory responses.

Monoclonal antiprothrombinase (3D4.3) prevents mortality from murine hepatitis virus (MHV-3) infection.

C5 deficiency and C5a or C5aR blockade protects against cerebral malaria

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114-124

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scholarly article

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10.1002/HEP.27114

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1

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60

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2014-05-28T00:00:00Z

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24604562

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