Amyloid-beta dynamics correlate with neurological status in the injured human brain - Wikidata - awgldk - TriplyDB

Amyloid-beta dynamics correlate with neurological status in the injured human brain

Amyloid-beta dynamics correlate with neurological status in the injured human brain

http://www.wikidata.org/entity/Q42241419

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The problem of pseudoreplication in neuroscientific studies: is it affecting your analysis?Chronic Traumatic Encephalopathy in Athletes: Progressive Tauopathy After Repetitive Head Injury Tau elevations in the brain extracellular space correlate with reduced amyloid-β levels and predict adverse clinical outcomes after severe traumatic brain injury Candidate mechanisms underlying the association between sleep-wake disruptions and Alzheimer's disease Impaired vascular-mediated clearance of brain amyloid beta in Alzheimer's disease: the role, regulation and restoration of LRP1 Consensus statement from the 2014 International Microdialysis Forum Cerebral microdialysis for protein biomarker monitoring in the neurointensive care setting - a technical approach Amyloid β precursor protein as a molecular target for amyloid β--induced neuronal degeneration in Alzheimer's disease Abnormal White Matter Blood-Oxygen-Level-Dependent Signals in Chronic Mild Traumatic Brain Injury.Emerging Roles for the Immune System in Traumatic Brain Injury Lead-induced accumulation of beta-amyloid in the choroid plexus: role of low density lipoprotein receptor protein-1 and protein kinase C Neurochemical profile of dementia pugilistica Molecular mechanisms of cognitive dysfunction following traumatic brain injury Lead exposure increases levels of β-amyloid in the brain and CSF and inhibits LRP1 expression in APP transgenic mice Increased beta-amyloid levels in the choroid plexus following lead exposure and the involvement of low-density lipoprotein receptor protein-1 Assessment of α-synuclein secretion in mouse and human brain parenchyma Alzheimer's therapeutics targeting amyloid beta 1-42 oligomers I: Abeta 42 oligomer binding to specific neuronal receptors is displaced by drug candidates that improve cognitive deficits Neurovascular pathways to neurodegeneration in Alzheimer's disease and other disorders Cortical hubs revealed by intrinsic functional connectivity: mapping, assessment of stability, and relation to Alzheimer's disease Basal cerebral metabolism may modulate the cognitive effects of Abeta in mild cognitive impairment: an example of brain reserve.Synaptic activity reduces intraneuronal Abeta, promotes APP transport to synapses, and protects against Abeta-related synaptic alterations Clinical review: neuromonitoring - an update.The amyloid cascade-inflammatory hypothesis of Alzheimer disease: implications for therapy.Amyloid seeds formed by cellular uptake, concentration, and aggregation of the amyloid-beta peptide.Acute dosing of latrepirdine (Dimebon), a possible Alzheimer therapeutic, elevates extracellular amyloid-beta levels in vitro and in vivo Neuroprotective pathways: lifestyle activity, brain pathology, and cognition in cognitively normal older adults.Multimodal techniques for diagnosis and prognosis of Alzheimer's disease Diurnal oscillation of CSF Aβ and other AD biomarkers.Lactic acid induces aberrant amyloid precursor protein processing by promoting its interaction with endoplasmic reticulum chaperone proteins.Genetics and molecular pathogenesis of sporadic and hereditary cerebral amyloid angiopathies Amyloid precursor protein secretases as therapeutic targets for traumatic brain injury.Cerebrospinal fluid biomarkers of Alzheimer's disease.Gene-environment interactions: lifetime cognitive activity, APOE genotype, and β-amyloid burden.Analyses of cerebral microdialysis in patients with traumatic brain injury: relations to intracranial pressure, cerebral perfusion pressure and catheter placement The Alzheimer's disease mitochondrial cascade hypothesis Picomolar amyloid-β peptides enhance spontaneous astrocyte calcium transients Synapses, synaptic activity and intraneuronal abeta in Alzheimer's disease.The intersection of amyloid beta and tau at synapses in Alzheimer's disease Cognition, glucose metabolism and amyloid burden in Alzheimer's disease.Dual effect of beta-amyloid on α7 and α4β2 nicotinic receptors controlling the release of glutamate, aspartate and GABA in rat hippocampus.

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Amyloid-beta dynamics correlate with neurological status in the injured human brain

http://www.wikidata.org/entity/Q42241419

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2008 nî lūn-bûn

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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2008年论文

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2008年论文

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name

Amyloid-beta dynamics correlate with neurological status in the injured human brain

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Amyloid-beta dynamics correlate with neurological status in the injured human brain

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type

label

Amyloid-beta dynamics correlate with neurological status in the injured human brain

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Amyloid-beta dynamics correlate with neurological status in the injured human brain

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prefLabel

Amyloid-beta dynamics correlate with neurological status in the injured human brain

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Amyloid-beta dynamics correlate with neurological status in the injured human brain

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Amyloid-beta dynamics correlate with neurological status in the injured human brain

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David L Brody

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Kate E Schwetye

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Michael L Spinner

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Nino Stocchetti

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Sandra Magnoni

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Thomas J Esparza

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P2860

Amyloid-beta protein dimers isolated directly from Alzheimer's brains impair synaptic plasticity and memory

Naturally secreted oligomers of amyloid beta protein potently inhibit hippocampal long-term potentiation in vivo

A specific amyloid-beta protein assembly in the brain impairs memory

Molecular, structural, and functional characterization of Alzheimer's disease: evidence for a relationship between default activity, amyloid, and memory

Natural oligomers of the amyloid-beta protein specifically disrupt cognitive function

APP processing and synaptic function

Assessment of coma and impaired consciousness. A practical scale

Intracerebral microdialysis in neurointensive care: the use of urea as an endogenous reference compound.

Beta amyloid protein deposition in the brain after severe head injury: implications for the pathogenesis of Alzheimer's disease.

Isolation and quantification of soluble Alzheimer's beta-peptide from biological fluids.

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321

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