Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
about
Primary erythromelalgia: a reviewBurning pain: axonal dysfunction in erythromelalgia.Network topology of NaV1.7 mutations in sodium channel-related painful disorders.Gain-of-Function Mutation W493R in the Epithelial Sodium Channel Allosterically Reconfigures Intersubunit Coupling.Analgesic Effects of GpTx-1, PF-04856264 and CNV1014802 in a Mouse Model of NaV1.7-Mediated Pain.
P2860
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
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2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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2015年论文
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2015年论文
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name
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@en
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@nl
type
label
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@en
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@nl
prefLabel
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@en
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@nl
P2860
P356
P1476
Erythromelalgia mutation Q875E Stabilizes the activated state of sodium channel Nav1.7.
@en
P2093
Andrias O O'Reilly
Theresa Stadler
P2860
P304
P356
10.1074/JBC.M114.605899
P407
P577
2015-01-09T00:00:00Z