Growth of Candida albicans cells on the physiologically relevant carbon source lactate affects their recognition and phagocytosis by immune cells.
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Adaptations of the Secretome of Candida albicans in Response to Host-Related Environmental ConditionsFungal immune evasion in a model host-pathogen interaction: Candida albicans versus macrophagesCharacterization of Virulence-Related Phenotypes in Candida Species of the CUG Clade.Modulation of phagosomal pH by Candida albicans promotes hyphal morphogenesis and requires Stp2p, a regulator of amino acid transportExperimental Models of Vaginal Candidiasis and Their Relevance to Human CandidiasisDressed to impress: impact of environmental adaptation on the Candida albicans cell wallInhibitors of the glyoxylate cycle enzyme ICL1 in Candida albicans for potential use as antifungal agentsIntegration of Posttranscriptional Gene Networks into Metabolic Adaptation and Biofilm Maturation in Candida albicansMitochondrial Complex I Is a Global Regulator of Secondary Metabolism, Virulence and Azole Sensitivity in FungiMitochondrial complex I bridges a connection between regulation of carbon flexibility and gastrointestinal commensalism in the human fungal pathogen Candida albicansMetabolism in fungal pathogenesis.Influence of culture media on biofilm formation by Candida species and response of sessile cells to antifungals and oxidative stress.The Rewiring of Ubiquitination Targets in a Pathogenic Yeast Promotes Metabolic Flexibility, Host Colonization and Virulence.Cell Wall Remodeling Enzymes Modulate Fungal Cell Wall Elasticity and Osmotic Stress Resistance.The Candida albicans ATO Gene Family Promotes Neutralization of the Macrophage PhagolysosomeN-Acetylglucosamine-Induced Cell Death in Candida albicans and Its Implications for Adaptive Mechanisms of Nutrient Sensing in Yeasts.A feast for Candida: Metabolic plasticity confers an edge for virulence.The Snf1-activating kinase Sak1 is a key regulator of metabolic adaptation and in vivo fitness of Candida albicans.Strain dependent variation of immune responses to A. fumigatus: definition of pathogenic speciesFungal cell wall dynamics and infection site microenvironments: signal integration and infection outcome.Robust Extracellular pH Modulation by Candida albicans during Growth in Carboxylic Acids.N-acetylglucosamine (GlcNAc)-inducible gene GIG2 is a novel component of GlcNAc metabolism in Candida albicans.Stress adaptation in a pathogenic fungus.Vulvovaginal Candida albicans infections: pathogenesis, immunity and vaccine prospects.Metabolism impacts upon Candida immunogenicity and pathogenicity at multiple levels.Evolution of regulatory networks in Candida glabrata: learning to live with the human host.Sfp1 and Rtg3 reciprocally modulate carbon source-conditional stress adaptation in the pathogenic yeast Candida albicansN-Acetylglucosamine Metabolism Promotes Survival of Candida albicans in the Phagosome.Modelling the host-pathogen interactions of macrophages and Candida albicans using Game Theory and dynamic optimization.The vaginal mycobiome: A contemporary perspective on fungi in women's health and diseasesCandida glabrata susceptibility to antifungals and phagocytosis is modulated by acetate.Ribosomal protein S6 phosphorylation is controlled by TOR and modulated by PKA in Candida albicans.Overview of carbon and nitrogen catabolite metabolism in the virulence of human pathogenic fungi.Lactate signalling regulates fungal β-glucan masking and immune evasion.The Genomic Landscape of the Fungus-Specific SWI/SNF Complex Subunit, Snf6, in Candida albicans.The carboxylic acid transporters Jen1 and Jen2 affect the architecture and fluconazole susceptibility of Candida albicans biofilm in the presence of lactate.Wor1 establishes opaque cell fate through inhibition of the general co-repressor Tup1 in Candida albicans.Macrophage Migration Is Impaired within Candida albicans Biofilms.Dynamic Fungal Cell Wall Architecture in Stress Adaptation and Immune Evasion.The Role of Candida albicans Transcription Factor RLM1 in Response to Carbon Adaptation.
P2860
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P2860
Growth of Candida albicans cells on the physiologically relevant carbon source lactate affects their recognition and phagocytosis by immune cells.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Growth of Candida albicans cel ...... phagocytosis by immune cells.
@en
Growth of Candida albicans cel ...... phagocytosis by immune cells.
@nl
type
label
Growth of Candida albicans cel ...... phagocytosis by immune cells.
@en
Growth of Candida albicans cel ...... phagocytosis by immune cells.
@nl
prefLabel
Growth of Candida albicans cel ...... phagocytosis by immune cells.
@en
Growth of Candida albicans cel ...... phagocytosis by immune cells.
@nl
P2860
P356
P1476
Growth of Candida albicans cel ...... d phagocytosis by immune cells
@en
P2093
Iuliana V Ene
Mihai G Netea
P2860
P304
P356
10.1128/IAI.01092-12
P407
P577
2012-10-31T00:00:00Z