Divergent regulation by growth factors and cytokines of 95 kDa and 72 kDa gelatinases and tissue inhibitors or metalloproteinases-1, -2, and -3 in rabbit aortic smooth muscle cells.
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Elevated levels of soluble CD163 in sera and fluids from rheumatoid arthritis patients and inhibition of the shedding of CD163 by TIMP-3Elevated levels of matrix metalloproteinase 9 and tissue inhibitor of metalloproteinase 1 during the acute phase of Kawasaki diseaseIL-1 beta induces proMMP-9 expression via c-Src-dependent PDGFR/PI3K/Akt/p300 cascade in rat brain astrocytesVascular smooth muscle cells in cerebral aneurysm pathogenesis.The C-terminal domains of ADAMTS-4 and ADAMTS-5 promote association with N-TIMP-3.Cortical spreading depression activates and upregulates MMP-9.Molecular mechanisms in intimal hyperplasia.The mechanisms of coronary restenosis: insights from experimental models.Therapeutic potential of matrix metalloproteinase inhibitors in atherosclerosis.Japanese encephalitis virus induces matrix metalloproteinase-9 in rat brain astrocytes via NF-κB signalling dependent on MAPKs and reactive oxygen species.Myocardial remodelling and matrix metalloproteinases in heart failure: turmoil within the interstitium.Doxycycline-a role in ocular surface repair.Japanese encephalitis virus induces matrix metalloproteinase-9 expression via a ROS/c-Src/PDGFR/PI3K/Akt/MAPKs-dependent AP-1 pathway in rat brain astrocytesThe genetic basis for altered blood vessel function in disease: large artery stiffeningCytokines and cardiovascular disease.Pericytes on the tumor vasculature: jekyll or hyde?Metalloproteinases and vulnerable atherosclerotic plaquesRole of matrix metalloproteinases (MMPs) and MMP inhibitors on intracranial aneurysms: a review article.Coronary artery bypass graft: why is the saphenous vein prone to intimal hyperplasia?Activation of protein kinase Czeta is essential for cytokine-induced metalloproteinase-1, -3, and -9 secretion from rabbit smooth muscle cells and inhibits proliferation.Naringin inhibits matrix metalloproteinase-9 expression and AKT phosphorylation in tumor necrosis factor-alpha-induced vascular smooth muscle cells.3,4,5-trihydroxybenzaldehyde from Geum japonicum has dual inhibitory effect on matrix metalloproteinase 9; inhibition of gelatinoytic activity as well as MMP-9 expression in TNF-alpha induced HASMC.A novel function of benzyl isothiocyanate in vascular smooth muscle cells: the role of ERK1/2, cell cycle regulation, and matrix metalloproteinase-9.Vascular Imaging of Matrix Metalloproteinase Activity as an Informative Preclinical Biomarker of Drug-induced Vascular Injury.Deficiency of the protein-tyrosine phosphatase DEP-1/PTPRJ promotes matrix metalloproteinase-9 expression in meningioma cells.Differential effects of tumor necrosis factor-α on matrix metalloproteinase-2 expression in human myometrial and uterine leiomyoma smooth muscle cells.Divergent effects of tissue inhibitor of metalloproteinase-1, -2, or -3 overexpression on rat vascular smooth muscle cell invasion, proliferation, and death in vitro. TIMP-3 promotes apoptosis.Assessment of the involvement of the macrophage migration inhibitory factor-glucocorticoid regulatory dyad in the expression of matrix metalloproteinase-2 during periodontitis.Inhibitory effect of chitooligosaccharides on matrix metalloproteinase-9 in human fibrosarcoma cells (HT1080).Nerve growth factor activation of Erk-1 and Erk-2 induces matrix metalloproteinase-9 expression in vascular smooth muscle cells.Matrix metalloproteinases increase very early during experimental focal cerebral ischemia.The oxygen free radicals control MMP-9 and transcription factors expression in the spontaneously hypertensive rat.Nuclear factor kappa B and matrix metalloproteinase induced receptor cleavage in the spontaneously hypertensive rat.Comparison of doxycycline and minocycline in the inhibition of VEGF-induced smooth muscle cell migration.Peroxisome proliferator-activated receptor gamma activators inhibit gene expression and migration in human vascular smooth muscle cells.Angiotensin II AT(2) receptor inhibits smooth muscle cell migration via fibronectin cell production and binding.Betulinic acid inhibits high glucose-induced vascular smooth muscle cells proliferation and migration.Acrolein-activated matrix metalloproteinase 9 contributes to persistent mucin production.Tranilast inhibits the proliferation, chemotaxis and tube formation of human microvascular endothelial cells in vitro and angiogenesis in vivo.Vascular smooth muscle cells and pericytes express MMP-1, MMP-9, TIMP-1 and type I procollagen in inflammatory bowel disease.
P2860
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P2860
Divergent regulation by growth factors and cytokines of 95 kDa and 72 kDa gelatinases and tissue inhibitors or metalloproteinases-1, -2, and -3 in rabbit aortic smooth muscle cells.
description
1996 nî lūn-bûn
@nan
1996年の論文
@ja
1996年論文
@yue
1996年論文
@zh-hant
1996年論文
@zh-hk
1996年論文
@zh-mo
1996年論文
@zh-tw
1996年论文
@wuu
1996年论文
@zh
1996年论文
@zh-cn
name
Divergent regulation by growth ...... it aortic smooth muscle cells.
@en
Divergent regulation by growth ...... it aortic smooth muscle cells.
@nl
type
label
Divergent regulation by growth ...... it aortic smooth muscle cells.
@en
Divergent regulation by growth ...... it aortic smooth muscle cells.
@nl
prefLabel
Divergent regulation by growth ...... it aortic smooth muscle cells.
@en
Divergent regulation by growth ...... it aortic smooth muscle cells.
@nl
P2093
P2860
P356
P1433
P1476
Divergent regulation by growth ...... it aortic smooth muscle cells.
@en
P2093
P2860
P304
P356
10.1042/BJ3150335
P407
P478
315 ( Pt 1)
P577
1996-04-01T00:00:00Z