Multiple functional domains of Enterococcus faecalis aggregation substance Asc10 contribute to endocarditis virulence.
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Peptide pheromone signaling in Streptococcus and EnterococcusEnterococcal Sex Pheromones: Evolutionary Pathways to Complex, Two-Signal SystemsVirulence Plasmids of Nonsporulating Gram-Positive PathogensRole of intracellular stochasticity in biofilm growth. Insights from population balance modelingBiological diversity of prokaryotic type IV secretion systems.Vaccination against Staphylococcus aureus pneumoniaEnterococcus faecalis endocarditis severity in rabbits is reduced by IgG Fabs interfering with aggregation substanceAcceleration of Enterococcus faecalis biofilm formation by aggregation substance expression in an ex vivo model of cardiac valve colonizationBistability versus bimodal distributions in gene regulatory processes from population balance.In vitro communities derived from oral and gut microbial floras inhibit the growth of bacteria of foreign origins.Friend turned foe: evolution of enterococcal virulence and antibiotic resistanceModified lactic acid bacteria detect and inhibit multiresistant enterococci.Biofilm growth alters regulation of conjugation by a bacterial pheromone.Use of recombinase-based in vivo expression technology to characterize Enterococcus faecalis gene expression during infection identifies in vivo-expressed antisense RNAs and implicates the protease Eep in pathogenesis.AhrC and Eep are biofilm infection-associated virulence factors in Enterococcus faecalis.Presence of virulence factors in Enterococcus faecalis and Enterococcus faecium susceptible and resistant to vancomycinVirulence Factors Associated with Enterococcus Faecalis Infective Endocarditis: A Mini Review.Model systems for the study of Enterococcal colonization and infection.Enterococcus faecalis readily colonizes the entire gastrointestinal tract and forms biofilms in a germ-free mouse model.Aortic Valve Damage for the Study of Left-Sided, Native Valve Infective Endocarditis in Rabbits.Enterococcus faecalis pCF10-encoded surface proteins PrgA, PrgB (aggregation substance) and PrgC contribute to plasmid transfer, biofilm formation and virulence.Enterococcus faecalis PrgJ, a VirB4-like ATPase, mediates pCF10 conjugative transfer through substrate binding.PrgU: a suppressor of sex pheromone toxicity in Enterococcus faecalis.Scientific Opinion on the maintenance of the list of QPS microorganisms intentionally added to food or feed (2009 update)Enterococcus faecalis Sex Pheromone cCF10 Enhances Conjugative Plasmid Transfer In Vivo.Mechanistic features of the enterococcal pCF10 sex pheromone response and the biology of Enterococcus faecalis in its natural habitat.PrgB Promotes Aggregation, Biofilm Formation, and Conjugation through DNA Binding and Compaction.
P2860
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P2860
Multiple functional domains of Enterococcus faecalis aggregation substance Asc10 contribute to endocarditis virulence.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Multiple functional domains of ...... ute to endocarditis virulence.
@en
Multiple functional domains of ...... ute to endocarditis virulence.
@nl
type
label
Multiple functional domains of ...... ute to endocarditis virulence.
@en
Multiple functional domains of ...... ute to endocarditis virulence.
@nl
prefLabel
Multiple functional domains of ...... ute to endocarditis virulence.
@en
Multiple functional domains of ...... ute to endocarditis virulence.
@nl
P2093
P2860
P356
P1476
Multiple functional domains of ...... ute to endocarditis virulence.
@en
P2093
Carol L Wells
Dawn A Manias
Gary M Dunny
Olivia N Chuang
Timothy J Tripp
P2860
P304
P356
10.1128/IAI.01034-08
P407
P577
2008-10-27T00:00:00Z