Release of cyclooxygenase-2 and lipoxin A4 from blood leukocytes in aspirin-exacerbated respiratory disease

about

Release of cyclooxygenase-2 and lipoxin A4 from blood leukocytes in aspirin-exacerbated respiratory disease

Item

http://www.wikidata.org/entity/Q42266554

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@en

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@nl

type

Item

label

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@en

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@nl

prefLabel

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@en

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@nl

P1476

Release of cyclooxygenase-2 an ...... xacerbated respiratory disease

@en

P2093

Ajnacska Rozsasi

http://www.w3.org/2001/XMLSchema#string

Akos Heinemann

http://www.w3.org/2001/XMLSchema#string

Tilman Keck

http://www.w3.org/2001/XMLSchema#string

P2860

Dynamics of COX-2 in nasal mucosa and nasal polyps from aspirin-tolerant and aspirin-intolerant patients with asthma

Prostaglandin, leukotriene, and lipoxin balance in chronic rhinosinusitis with and without nasal polyposis

Expression of cyclooxygenase and lipoxygenase enzymes in nasal polyps of aspirin-sensitive and aspirin-tolerant patients

Lipoxin A4 levels in asthma: relation with disease severity and aspirin sensitivity

Factors driving the aspirin exacerbated respiratory disease phenotype.

Prominent role of IFN-γ in patients with aspirin-exacerbated respiratory disease.

Interleukin-4 in the Generation of the AERD Phenotype: Implications for Molecular Mechanisms Driving Therapeutic Benefit of Aspirin Desensitization.

Signal transduction pathways (MAPKs, NF-κB, and C/EBP) regulating COX-2 expression in nasal fibroblasts from asthma patients with aspirin intolerance.

Molecular mechanisms underlying chemopreventive activities of anti-inflammatory phytochemicals: down-regulation of COX-2 and iNOS through suppression of NF-kappa B activation.

EP4 receptor stimulation down-regulates human eosinophil function.

P304

158-163

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.2500/AR.2016.7.0172

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-01-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28107149

http://www.w3.org/2001/XMLSchema#string

P932

5244273

http://www.w3.org/2001/XMLSchema#string