Wnt 10b activates the CCN2 promoter in NIH 3T3 fibroblasts through the Smad response element.

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The Notch ligand Delta-like 1 integrates inputs from TGFbeta/Activin and Wnt pathways.Will o' the wisp: CCN4 as a novel molecular target in osteoarthritis.

P2860

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Wnt 10b activates the CCN2 promoter in NIH 3T3 fibroblasts through the Smad response element.

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http://www.wikidata.org/entity/Q42274400

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2009 nî lūn-bûn

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2009年の論文

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2009年論文

@yue

2009年論文

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2009年論文

@zh-hk

2009年論文

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@zh-tw

2009年论文

@wuu

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2009年论文

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Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@en

Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@nl

type

Item

label

Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@en

Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@nl

prefLabel

Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@en

Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@nl

P1476

Wnt 10b activates the CCN2 pro ...... ugh the Smad response element.

@en

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Andrew Leask

http://www.w3.org/2001/XMLSchema#string

Shaoqiong Chen

http://www.w3.org/2001/XMLSchema#string

P2860

Tumor necrosis factor alpha suppresses the induction of connective tissue growth factor by transforming growth factor-beta in normal and scleroderma fibroblasts

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The Wnt/Ca2+ pathway: a new vertebrate Wnt signaling pathway takes shape

Smad3-dependent nuclear translocation of beta-catenin is required for TGF-beta1-induced proliferation of bone marrow-derived adult human mesenchymal stem cells.

Wnt signaling: is the party in the nucleus?

Signaling cross-talk between TGF-beta/BMP and other pathways.

The murine gastrin promoter is synergistically activated by transforming growth factor-beta/Smad and Wnt signaling pathways.

The effects of myostatin on adipogenic differentiation of human bone marrow-derived mesenchymal stem cells are mediated through cross-communication between Smad3 and Wnt/beta-catenin signaling pathways.

The gene expression profile induced by Wnt 3a in NIH 3T3 fibroblasts.

CTGF and SMADs, maintenance of scleroderma phenotype is independent of SMAD signaling.

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s12079-009-0049-3

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57-59

http://www.w3.org/2001/XMLSchema#string

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scholarly article

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10.1007/S12079-009-0049-3

http://www.w3.org/2001/XMLSchema#string

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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2009-03-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

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24263742

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19353303

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P932

2686755

http://www.w3.org/2001/XMLSchema#string

Wnt 10b activates the CCN2 promoter in NIH 3T3 fibroblasts through the Smad response element.

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P2860

P2860

Wnt 10b activates the CCN2 promoter in NIH 3T3 fibroblasts through the Smad response element.

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type

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P2093

P2860

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P356

P433

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P577

P5875

P698

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P1476

P2093

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P356

P433

P478

P577

P5875

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