Chimeric Peptide Tat-HA-NR2B9c Improves Regenerative Repair after Transient Global Ischemia.

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Chimeric Peptide Tat-HA-NR2B9c Improves Regenerative Repair after Transient Global Ischemia.

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2017 nî lūn-bûn

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2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

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2017年论文

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2017年论文

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name

Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

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Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

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Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

@en

Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

@nl

prefLabel

Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

@en

Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

@nl

P1476

Chimeric Peptide Tat-HA-NR2B9c ...... ter Transient Global Ischemia.

@en

P2093

Hai-Hui Zhou

http://www.w3.org/2001/XMLSchema#string

Hai-Xia Zhang

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Jin-Ping Zhang

http://www.w3.org/2001/XMLSchema#string

Li Zhang

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Wei-Hong Ge

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P2860

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DAPK1 interaction with NMDA receptor NR2B subunits mediates brain damage in stroke

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NMDA receptors activated by subventricular zone astrocytic glutamate are critical for neuroblast survival prior to entering a synaptic network

Chronic glucocorticoid exposure activates BK-NLRP1 signal involving in hippocampal neuron damage.

Mechanisms underlying hypoxia-induced neuronal apoptosis.

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509

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P31

scholarly article

P356

10.3389/FNEUR.2017.00509

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P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-09-26T00:00:00Z

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29018405

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5622973

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