Cutting edge: in the absence of TGF-β signaling in T cells, fewer CD103+ regulatory T cells develop, but exuberant IFN-γ production renders mice more susceptible to helminth infection.
about
Immunity to the model intestinal helminth parasite Heligmosomoides polygyrusInnate and adaptive type 2 immune cell responses in genetically controlled resistance to intestinal helminth infectionModification of hemodynamic and immune responses to exposure with a weak antigen by the expression of a hypomorphic BMPR2 geneRegulatory T cells control NK cells in an insulitic lesion by depriving them of IL-2.Helminths and their implication in sepsis - a new branch of their immunomodulatory behaviour?Loss of the TGFβ-activating integrin αvβ8 on dendritic cells protects mice from chronic intestinal parasitic infection via control of type 2 immunity.Systemic impact of intestinal helminth infections.The Oral Tolerance as a Complex Network PhenomenonTGF-β in tolerance, development and regulation of immunityDifferential control of Helios(+/-) Treg development by monocyte subsets through disparate inflammatory cytokinesSystems biologic analysis of T regulatory cells genetic pathways in murine primary biliary cirrhosis.Th9 cells: differentiation and disease.Expansion of Foxp3(+) T-cell populations by Candida albicans enhances both Th17-cell responses and fungal dissemination after intravenous challenge.IL-9 and Th9 in parasite immunity.Cultivation of Heligmosomoides polygyrus: an immunomodulatory nematode parasite and its secreted productsSuccessful immunotherapy of autoimmune cholangitis by adoptive transfer of forkhead box protein 3(+) regulatory T cells.IL-25 exhibits disparate roles during Th2-cell differentiation versus effector function.Immune modulation of Th1, Th2, and T-reg transcriptional factors differing from cytokine levels in Schistosoma japonicum infection.TGF-β mimic proteins form an extended gene family in the murine parasite Heligmosomoides polygyrus.
P2860
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P2860
Cutting edge: in the absence of TGF-β signaling in T cells, fewer CD103+ regulatory T cells develop, but exuberant IFN-γ production renders mice more susceptible to helminth infection.
description
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name
Cutting edge: in the absence o ...... eptible to helminth infection.
@en
Cutting edge: in the absence o ...... eptible to helminth infection.
@nl
type
label
Cutting edge: in the absence o ...... eptible to helminth infection.
@en
Cutting edge: in the absence o ...... eptible to helminth infection.
@nl
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Cutting edge: in the absence o ...... eptible to helminth infection.
@en
Cutting edge: in the absence o ...... eptible to helminth infection.
@nl
P2860
P356
P1476
Cutting edge: in the absence o ...... eptible to helminth infection.
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P2093
Lisa A Reynolds
Rick M Maizels
P2860
P304
P356
10.4049/JIMMUNOL.1200991
P407
P577
2012-06-29T00:00:00Z