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Obstructive sleep apnoea pathogenesis from mild to severe: Is it all the same?Arousal from sleep does not lead to reduced dilator muscle activity or elevated upper airway resistance on return to sleep in healthy individualsThe pharyngeal lumen: both length and size matter.Ventilatory control and airway anatomy in obstructive sleep apnea.Obstructive sleep apnea and aging effects on macrovascular and microcirculatory function.Discharge patterns of human genioglossus motor units during arousal from sleep.A secondary reflex suppression phase is present in genioglossus but not tensor palatini in response to negative upper airway pressure.Motor unit recruitment in human genioglossus muscle in response to hypercapnia.Enhanced upper-airway muscle responsiveness is a distinct feature of overweight/obese individuals without sleep apnea.Effect of oxygen in obstructive sleep apnea: role of loop gain.Activation of the upper airway dilator muscle genioglossus during sleep is largely dependent on an interaction between chemical drive and mechanoreceptor feedback.Effect of CPAP on intrinsic PEEP, inspiratory effort, and lung volume in severe stable COPD.Recent advances in understanding the pathogenesis of obstructive sleep apnea.Sensorimotor function of the upper-airway muscles and respiratory sensory processing in untreated obstructive sleep apnea.An Integrative Model of Physiological Traits Can be Used to Predict Obstructive Sleep Apnea and Response to Non Positive Airway Pressure Therapy.Crossed motor innervation of the base of human tongueNeurogenic changes in the upper airway of patients with obstructive sleep apnea.Discharge patterns of human tensor palatini motor units during sleep onset.Unwarranted administration of acetylcholinesterase inhibitors can impair genioglossus and diaphragm muscle function.Effect of expiratory positive airway pressure on sleep disordered breathingThe influence of aging on pharyngeal collapsibility during sleep.Central sleep apnea: Pathophysiology and treatment.Common drive to the upper airway muscle genioglossus during inspiratory loadingUpper Airway Collapsibility (Pcrit) and Pharyngeal Dilator Muscle Activity are Sleep Stage DependentMild Airflow Limitation during N2 Sleep Increases K-complex Frequency and Slows Electroencephalographic Activity.Upper airway myopathy is not important in the pathophysiology of obstructive sleep apneaDifferential effects of isoflurane and propofol on upper airway dilator muscle activity and breathing.The influence of end-expiratory lung volume on measurements of pharyngeal collapsibility.Lung volume and continuous positive airway pressure requirements in obstructive sleep apneaDefining phenotypic causes of obstructive sleep apnea. Identification of novel therapeutic targets.Pentobarbital dose-dependently increases respiratory genioglossus muscle activity while impairing diaphragmatic function in anesthetized ratsNasal Resistance Is Elevated in People with Tetraplegia and Is Reduced by Topical Sympathomimetic Administration.Arousal Intensity is a Distinct Pathophysiological Trait in Obstructive Sleep Apnea.The influence of obstructive sleep apnea and gender on genioglossus activity during rapid eye movement sleep.Adult obstructive sleep apnoea.Physiological mechanisms of upper airway hypotonia during REM sleep.Sleep and cardiovascular regulation.Discharge properties of upper airway motor units during wakefulness and sleep.Therapeutic CPAP Level Predicts Upper Airway Collapsibility in Patients With Obstructive Sleep Apnea.Physiology of Arousal in OSA and Potential Impacts for Sedative Treatment.
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hulumtuese
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wetenschapper
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հետազոտող
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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Amy S. Jordan
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