MMI-0100 Inhibits Cardiac Fibrosis in a Mouse Model Overexpressing Cardiac Myosin Binding Protein C.

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MMI-0100 Inhibits Cardiac Fibrosis in a Mouse Model Overexpressing Cardiac Myosin Binding Protein C.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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MMI-0100 Inhibits Cardiac Fibr ...... diac Myosin Binding Protein C.

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MMI-0100 Inhibits Cardiac Fibr ...... diac Myosin Binding Protein C.

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MMI-0100 Inhibits Cardiac Fibr ...... diac Myosin Binding Protein C.

@en

MMI-0100 Inhibits Cardiac Fibr ...... diac Myosin Binding Protein C.

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MMI-0100 Inhibits Cardiac Fibr ...... diac Myosin Binding Protein C.

@en

MMI-0100 Inhibits Cardiac Fibr ...... diac Myosin Binding Protein C.

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P1476

MMI-0100 Inhibits Cardiac Fibr ...... rdiac Myosin Binding Protein C

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P2093

Bidur Bhandary

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Cynthia Lander

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Hanna Osinska

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James Gulick

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Jeanne James

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Jeffrey Robbins

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Kritton Shay-Winkler

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Na Xu

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Qinghang Meng

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P2860

The myofibroblast: one function, multiple origins

The role of the myofibroblast in tumor stroma remodeling

MMI-0100 inhibits cardiac fibrosis in myocardial infarction by direct actions on cardiomyocytes and fibroblasts via MK2 inhibition.

Getting to the heart of the matter: new insights into cardiac fibrosis.

Cardiomyocyte-Specific Human Bcl2-Associated Anthanogene 3 P209L Expression Induces Mitochondrial Fragmentation, Bcl2-Associated Anthanogene 3 Haploinsufficiency, and Activates p38 Signaling

Cardiac myosin binding protein C phosphorylation is cardioprotective

TGF-beta signaling and the fibrotic response

A Pleiotropic RNA-Binding Protein Controls Distinct Cell Cycle Checkpoints to Drive Resistance of p53-Defective Tumors to Chemotherapy

Genetic lineage tracing defines myofibroblast origin and function in the injured heart.

Hypertrophic cardiomyopathy: a systematic review.

P31

scholarly article

P356

10.1161/JAHA.117.006590

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P433

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P478

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P50

Monte Willis

P577

2017-09-04T00:00:00Z

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P698

28871043

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P921

overexpression

P932

5634300

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