MMP9 regulates the cellular response to inflammation after skeletal injury. - Wikidata - awgldk - TriplyDB

MMP9 regulates the cellular response to inflammation after skeletal injury.

MMP9 regulates the cellular response to inflammation after skeletal injury.

http://www.wikidata.org/entity/Q42383995

about

The multifaceted role of the vasculature in endochondral fracture repair Unraveling macrophage contributions to bone repair Biochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9): the next decade Tendon regeneration and scar formation: The concept of scarless healing Bioactive lipid coating of bone allografts directs engraftment and fate determination of bone marrow-derived cells in rat GFP chimeras The shared crosstalk of multiple pathways involved in the inflammation between rheumatoid arthritis and coronary artery disease based on a digital gene expression profile Association between four MMP-9 polymorphisms and breast cancer risk: a meta-analysis.The Crosstalk of Pathways Involved in Immune Response Maybe the Shared Molecular Basis of Rheumatoid Arthritis and Type 2 Diabetes Low-Dose and Short-Duration Matrix Metalloproteinase 9 Inhibition Does Not Affect Adhesion Formation during Murine Flexor Tendon Healing.Macrophages: Their Emerging Roles in Bone.Delayed bone regeneration is linked to chronic inflammation in murine muscular dystrophy.Periosteum: biology and applications in craniofacial bone regeneration.Cartilage to bone transformation during fracture healing is coordinated by the invading vasculature and induction of the core pluripotency genes.Effects of naringin on physical fatigue and serum MMP-9 concentration in female rats.Ablation of Y1 receptor impairs osteoclast bone-resorbing activity.Anguillicola crassus infection affects mRNA expression levels in gas gland tissue of European yellow and silver eel.Harnessing Endogenous Cellular Mechanisms for Bone Repair.Microarray gene expression of periosteum in spontaneous bone regeneration of mandibular segmental defects.Identification of key genes influenced by fixation stability in early fracture hematoma and elucidation of their roles in fracture healing.Role of muscle stem cells during skeletal regeneration.Biomimetic Approaches for Bone Tissue Engineering.Microenvironmental Regulation of Chondrocyte Plasticity in Endochondral Repair-A New Frontier for Developmental Engineering.The Antiosteoporosis Effects of Zhuanggu Guanjie Pill and

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MMP9 regulates the cellular response to inflammation after skeletal injury.

http://www.wikidata.org/entity/Q42383995

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

MMP9 regulates the cellular response to inflammation after skeletal injury.

@en

MMP9 regulates the cellular response to inflammation after skeletal injury.

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type

label

MMP9 regulates the cellular response to inflammation after skeletal injury.

@en

MMP9 regulates the cellular response to inflammation after skeletal injury.

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prefLabel

MMP9 regulates the cellular response to inflammation after skeletal injury.

@en

MMP9 regulates the cellular response to inflammation after skeletal injury.

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J.BONE.2012.09.018

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P1476

MMP9 regulates the cellular response to inflammation after skeletal injury.

@en

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Chuanyong Lu

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Céline Colnot

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Diane Hu

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Frank Yang

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Jeffrey Lang

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Ralph Marcucio

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Shirley Lieu

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Theodore Miclau

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Xiaodong Wang

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Yan Yiu Yu

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P2860

MMP-9/gelatinase B is a key regulator of growth plate angiogenesis and apoptosis of hypertrophic chondrocytes

Current insights on the regenerative potential of the periosteum: molecular, cellular, and endogenous engineering approaches

Beta-catenin signaling plays a disparate role in different phases of fracture repair: implications for therapy to improve bone healing

Role of matrix metalloproteinase 13 in both endochondral and intramembranous ossification during skeletal regeneration

Mechanical stimulation alters tissue differentiation and molecular expression during bone healing

Matrix metalloproteinases: effectors of development and normal physiology

Fracture healing is accelerated in the absence of the adaptive immune system.

Cellular basis for age-related changes in fracture repair

Complementary interplay between matrix metalloproteinase-9, vascular endothelial growth factor and osteoclast function drives endochondral bone formation

Activation of the Hh pathway in periosteum-derived mesenchymal stem cells induces bone formation in vivo: implication for postnatal bone repair.

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10.1016/J.BONE.2012.09.018

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1

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