Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

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Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

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http://www.wikidata.org/entity/Q42389546

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2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

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Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

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Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

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Item

label

Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

@en

Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

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Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

@en

Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

@nl

P1476

Prorenin Receptor, a Necessary Component in Urine Concentration Mechanism.

@en

P2093

Boye L Jensen

http://www.w3.org/2001/XMLSchema#string

P2860

Pivotal role of the renin/prorenin receptor in angiotensin II production and cellular responses to renin

Nephron-specific deletion of the prorenin receptor causes a urine concentration defect.

Dehydration-induced increase in aquaporin-2 protein abundance is blocked by nonsteroidal anti-inflammatory drugs.

COX-2 mediates angiotensin II-induced (pro)renin receptor expression in the rat renal medulla

Vasopressin-independent targeting of aquaporin-2 by selective E-prostanoid receptor agonists alleviates nephrogenic diabetes insipidus.

Prostaglandin E-prostanoid4 receptor mediates angiotensin II-induced (pro)renin receptor expression in the rat renal medulla.

Disruption of prostaglandin E2 receptor EP4 impairs urinary concentration via decreasing aquaporin 2 in renal collecting ducts.

A selective EP4 PGE2 receptor agonist alleviates disease in a new mouse model of X-linked nephrogenic diabetes insipidus.

(Pro)renin receptor is required for prorenin-dependent and -independent regulation of vacuolar H⁺-ATPase activity in MDCK.C11 collecting duct cells.

P304

2919-2921

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

editorial

P356

10.1681/ASN.2016030344

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-04-20T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27098238

http://www.w3.org/2001/XMLSchema#string

P932

5042682

http://www.w3.org/2001/XMLSchema#string