RANKL expression in periodontal disease: where does RANKL come from? - Wikidata - awgldk - TriplyDB

RANKL expression in periodontal disease: where does RANKL come from?

RANKL expression in periodontal disease: where does RANKL come from?

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OCLI-023, a Novel Pyrimidine Compound, Suppresses Osteoclastogenesis In Vitro and Alveolar Bone Resorption In Vivo Genes related to inflammation and bone loss process in periodontitis suggested by bioinformatics methods.Thymosin Beta-4 Suppresses Osteoclastic Differentiation and Inflammatory Responses in Human Periodontal Ligament Cells.A Modified Glycosaminoglycan, GM-0111, Inhibits Molecular Signaling Involved in Periodontitis.Osteoblast Lineage Cells Play an Essential Role in Periodontal Bone Loss Through Activation of Nuclear Factor-Kappa B Expression of SOFAT by T- and B-lineage cells may contribute to bone loss Osteoclasts-Key Players in Skeletal Health and Disease.Influence of Type 2 Diabetes on Prevalence of Key Periodontal Pathogens, Salivary Matrix Metalloproteinases, and Bone Remodeling Markers in Sudanese Adults with and without Chronic Periodontitis.The Cell Surface Markers Expression in Postmenopausal Women and Relation to Obesity and Bone Status.Tumor necrosis factor-α antagonist diminishes osteocytic RANKL and sclerostin expression in diabetes rats with periodontitis.Kava-241 reduced periodontal destruction in a collagen antibody primed Porphyromonas gingivalis model of periodontitis.Host defense against oral microbiota by bone-damaging T cells.

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RANKL expression in periodontal disease: where does RANKL come from?

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2014 nî lūn-bûn

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RANKL expression in periodontal disease: where does RANKL come from?

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RANKL expression in periodontal disease: where does RANKL come from?

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RANKL expression in periodontal disease: where does RANKL come from?

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RANKL expression in periodontal disease: where does RANKL come from?

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Weibin Sun

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Wenlei Wu

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Creative Commons Attribution 3.0 Unported

P2860

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Activated human T cells express alternative mRNA transcripts encoding a secreted form of RANKL

Modulation of osteoclast differentiation and function by the new members of the tumor necrosis factor receptor and ligand families

Matrix-embedded cells control osteoclast formation

Evidence for osteocyte regulation of bone homeostasis through RANKL expression

Activation of murine macrophages by lipoprotein and lipooligosaccharide of Treponema denticola.

CD4(+) T cells and the proinflammatory cytokines gamma interferon and interleukin-6 contribute to alveolar bone loss in mice

Involvement of T-lymphocytes in periodontal disease and in direct and indirect induction of bone resorption.

Physiological functions of osteoblast lineage and T cell-derived RANKL in bone homeostasis.

Functional human T-cell immunity and osteoprotegerin ligand control alveolar bone destruction in periodontal infection.

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731039

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10.1155/2014/731039

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2014

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261521841

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24719884

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regulation of gene expression

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3955606

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